S A Shurtleff
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Explore the profile of S A Shurtleff including associated specialties, affiliations and a list of published articles.
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38
Citations
1337
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Recent Articles
11.
Bonilla M, Moreno N, Marina N, deReyes G, Shurtleff S, Downing J, et al.
J Pediatr Hematol Oncol
. 2000 Dec;
22(6):495-501.
PMID: 11132215
Purpose: To improve outcome and study biology of childhood acute lymphoblastic leukemia (ALL) in El Salvador. Patients And Methods: Between January 1994 and December 1996, 153 children of El Salvador...
12.
Raynaud S, Dastugue N, Zoccola D, Shurtleff S, Mathew S, Raimondi S
Leukemia
. 1999 Sep;
13(9):1325-30.
PMID: 10482981
The t(12;21)(p13;q22) is a cryptic abnormality observed in 25% of children with B-lineage acute lymphoblastic leukemia (ALL), associated with a favorable prognosis. To determine whether specific cytogenetic abnormalities accompany the...
13.
Scurto P, Hsu Rocha M, Kane J, Williams W, Haney D, Conn W, et al.
Leukemia
. 1998 Dec;
12(12):1994-2005.
PMID: 9844930
Modern therapy for pediatric acute lymphoblastic leukemia (ALL) is based on the principle of risk stratification. One of the most important laboratory features used to accurately risk stratify patients is...
14.
Raimondi S, Shurtleff S, Downing J, Rubnitz J, Mathew S, Hancock M, et al.
Blood
. 1997 Dec;
90(11):4559-66.
PMID: 9373267
Although abnormalities involving the short arm of chromosome 12 (12p) are one of the most frequently observed rearrangements in childhood acute lymphoblastic leukemia (ALL), little is known about the frequency...
15.
Antillon F, Kaste S, Jenkins J, Shurtleff S, Merchant T, Downing J, et al.
J Pediatr Hematol Oncol
. 1997 Nov;
19(5):473-6.
PMID: 9329474
Purpose: Although rare, second malignant neoplasms (SMNs) are a devastating consequence of successful treatment of childhood cancer. The 15-year estimated risk of developing a second malignant neoplasm after treatment of...
16.
Venkateswaran L, Jenkins J, Kaste S, Shurtleff S, Downing J, Pappo A
J Pediatr Hematol Oncol
. 1997 Mar;
19(2):172-5.
PMID: 9149753
Purpose: Recently recognized as a distinct clinicopathologic entity, desmoplastic small round-cell tumors typically affect young men. These aggressive tumors usually arise in the abdomen; other sites of primary disease have...
17.
Rubnitz J, Downing J, Pui C, Shurtleff S, Raimondi S, Evans W, et al.
J Clin Oncol
. 1997 Mar;
15(3):1150-7.
PMID: 9060558
Purpose: TEL gene rearrangements due to the 12;21 chromosomal translocation are the most common molecular genetic abnormality in childhood acute lymphoblastic leukemia (ALL), occurring in approximately 25% of cases with...
18.
Kane J, Head D, Balazs L, Hulshof M, Motroni T, Raimondi S, et al.
Leukemia
. 1996 Aug;
10(8):1296-302.
PMID: 8709634
Acute promyelocytic leukemia (APL) is characterized cytogenetically by the t(15;17)(q22;q11-21) translocation. To compare molecular events among pediatric and adult APL cases, we designed two sets of oligonucleotide primers using published...
19.
Liang D, Chou T, Chen J, Shurtleff S, Rubnitz J, Downing J, et al.
Leukemia
. 1996 Jun;
10(6):991-3.
PMID: 8667657
Despite its rarity by routine karyotypic analysis, cryptic t(12;21)(p12-13;q22) translocation leading to TEL/AML1 fusion has been recognized as the most frequent genetic rearrangement in childhood acute lymphoblastic leukemia (ALL) in...
20.
Rubnitz J, Behm F, Pinheiro R, Carroll A, Raimondi S, Shurtleff S, et al.
Blood
. 1996 Jun;
87(11):4804-8.
PMID: 8639852
MLL is fused to ENL or ELL in acute leukemias that contain t(ll;19)(q23;p13). Although ENL and ELL localize to chromosome 19, bands p13.3 and p13.1, respectively, these breakpoints are not...