Richard F Wooster
Overview
Explore the profile of Richard F Wooster including associated specialties, affiliations and a list of published articles.
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7
Citations
736
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0
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Recent Articles
1.
Ramos-Montoya A, Lamb A, Russell R, Carroll T, Jurmeister S, Galeano-Dalmau N, et al.
EMBO Mol Med
. 2014 Apr;
6(5):651-61.
PMID: 24737870
Castrate-resistant prostate cancer (CRPC) is poorly characterized and heterogeneous and while the androgen receptor (AR) is of singular importance, other factors such as c-Myc and the E2F family also play...
2.
Billiard J, Dennison J, Briand J, Annan R, Chai D, Colon M, et al.
Cancer Metab
. 2013 Nov;
1(1):19.
PMID: 24280423
Background: Most normal cells in the presence of oxygen utilize glucose for mitochondrial oxidative phosphorylation. In contrast, many cancer cells rapidly convert glucose to lactate in the cytosol, a process...
3.
van den Heuvel A, Jing J, Wooster R, Bachman K
Cancer Biol Ther
. 2012 Aug;
13(12):1185-94.
PMID: 22892846
One of the hallmarks of cancer is metabolic deregulation. Many tumors display increased glucose uptake and breakdown through the process of aerobic glycolysis, also known as the Warburg effect. Less...
4.
5.
Heiser L, Sadanandam A, Kuo W, Benz S, Goldstein T, Ng S, et al.
Proc Natl Acad Sci U S A
. 2011 Oct;
109(8):2724-9.
PMID: 22003129
Breast cancers are comprised of molecularly distinct subtypes that may respond differently to pathway-targeted therapies now under development. Collections of breast cancer cell lines mirror many of the molecular subtypes...
6.
Wood K, Lad L, Luo L, Qian X, Knight S, Nevins N, et al.
Proc Natl Acad Sci U S A
. 2010 Feb;
107(13):5839-44.
PMID: 20167803
Centromere-associated protein-E (CENP-E) is a kinetochore-associated mitotic kinesin that is thought to function as the key receptor responsible for mitotic checkpoint signal transduction after interaction with spindle microtubules. We have...
7.
Heiser L, Wang N, Talcott C, Laderoute K, Knapp M, Guan Y, et al.
Genome Biol
. 2009 Mar;
10(3):R31.
PMID: 19317917
Background: Cancer is a heterogeneous disease resulting from the accumulation of genetic defects that negatively impact control of cell division, motility, adhesion and apoptosis. Deregulation in signaling along the EgfR-MAPK...