Raul H Bortolin
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Explore the profile of Raul H Bortolin including associated specialties, affiliations and a list of published articles.
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19
Citations
129
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Recent Articles
1.
Lu F, Liou C, Ma Q, Wu Z, Xue B, Xia Y, et al.
Nat Biomed Eng
. 2024 Sep;
PMID: 39237710
Cardiomyocytes derived from human induced pluripotent stem cells (hiPSC-CMs) lack nanoscale structures essential for efficient excitation-contraction coupling. Such nanostructures, known as dyads, are frequently disrupted in heart failure. Here we...
2.
Sweat M, Cao Y, Zhang X, Burnicka-Turek O, Perez-Cervantes C, Kulandaisamy A, et al.
Nat Cardiovasc Res
. 2024 Aug;
2(11):1095.
PMID: 39196102
No abstract available.
3.
Bortolin R, Nawar F, Park C, Trembley M, Prondzynski M, Sweat M, et al.
Circulation
. 2024 Aug;
150(15):1199-1210.
PMID: 39155863
Background: Calmodulinopathies are rare inherited arrhythmia syndromes caused by dominant heterozygous variants in , , or , which each encode the identical CaM (calmodulin) protein. We hypothesized that antisense oligonucleotide...
4.
Prondzynski M, Berkson P, Trembley M, Tharani Y, Shani K, Bortolin R, et al.
Nat Commun
. 2024 Jul;
15(1):5929.
PMID: 39009604
Human iPSC-derived cardiomyocytes (hiPSC-CMs) have proven invaluable for cardiac disease modeling and regeneration. Challenges with quality, inter-batch consistency, cryopreservation and scale remain, reducing experimental reproducibility and clinical translation. Here, we...
5.
Prondzynski M, Bortolin R, Berkson P, Trembley M, Shani K, Sweat M, et al.
bioRxiv
. 2024 Mar;
PMID: 38464269
In the last decade human iPSC-derived cardiomyocytes (hiPSC-CMs) proved to be valuable for cardiac disease modeling and cardiac regeneration, yet challenges with scale, quality, inter-batch consistency, and cryopreservation remain, reducing...
6.
Sweat M, Cao Y, Zhang X, Burnicka-Turek O, Perez-Cervantes C, Arulsamy K, et al.
Nat Cardiovasc Res
. 2024 Feb;
2(10):881-898.
PMID: 38344303
Understanding how the atrial and ventricular heart chambers maintain distinct identities is a prerequisite for treating chamber-specific diseases. Here, we selectively knocked out (KO) the transcription factor in the atrial...
7.
Sweat M, Cao Y, Zhang X, Burnicka-Turek O, Perez-Cervantes C, Akerberg B, et al.
bioRxiv
. 2023 May;
PMID: 37131696
Understanding how the atrial and ventricular chambers of the heart maintain their distinct identity is a prerequisite for treating chamber-specific diseases. Here, we selectively inactivated the transcription factor in the...
8.
Castelli E, de Castro M, Naslavsky M, Scliar M, Silva N, Pereira R, et al.
Front Immunol
. 2022 Nov;
13:975918.
PMID: 36389712
Background: Although aging correlates with a worse prognosis for Covid-19, super elderly still unvaccinated individuals presenting mild or no symptoms have been reported worldwide. Most of the reported genetic variants...
9.
de Melo T, de Souza K, Ururahy M, Bortolin R, Bezerra J, de Oliveira Galvao M, et al.
J Paediatr Child Health
. 2022 Jan;
58(6):996-1000.
PMID: 35006634
Aim: This study aimed to evaluate the association of toll-like receptor (TLR) inflammatory cascade with the development of diabetic kidney disease (DKD) in children and adolescents with type 1 diabetes...
10.
Castelli E, de Castro M, Naslavsky M, Scliar M, Silva N, Andrade H, et al.
Front Immunol
. 2021 Oct;
12:742881.
PMID: 34650566
Despite the high number of individuals infected by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) who develop coronavirus disease 2019 (COVID-19) symptoms worldwide, many exposed individuals remain asymptomatic and/or uninfected...