Raphaella W L So
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Explore the profile of Raphaella W L So including associated specialties, affiliations and a list of published articles.
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9
Citations
235
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Recent Articles
1.
Stykel M, Siripala S, Soubeyrand E, Coackley C, Lu P, Camargo S, et al.
Cell Rep
. 2025 Jan;
44(1):115178.
PMID: 39772392
Loss of dopaminergic neurons in Parkinson's disease (PD) is preceded by loss of synaptic dopamine (DA) and accumulation of proteinaceous aggregates. Linking these deficits is critical to restoring DA signaling...
2.
So R, Amano G, Stuart E, Ebrahim Amini A, Aguzzi A, Collingridge G, et al.
PLoS Pathog
. 2024 Sep;
20(9):e1012517.
PMID: 39264912
The cellular prion protein, PrPC, has been postulated to function as a receptor for α-synuclein, potentially facilitating cell-to-cell spreading and/or toxicity of α-synuclein aggregates in neurodegenerative disorders such as Parkinson's...
3.
Germain K, So R, DiGiovanni L, Watts J, Bandsma R, Kim P
Nat Commun
. 2024 Jan;
15(1):375.
PMID: 38195640
Selective autophagy is an essential process to maintain cellular homeostasis through the constant recycling of damaged or superfluous components. Over a dozen selective autophagy pathways mediate the degradation of diverse...
4.
Lau H, Martinez-Valbuena I, So R, Mehra S, Silver N, Mao A, et al.
Acta Neuropathol Commun
. 2023 May;
11(1):72.
PMID: 37138318
Unique strains of α-synuclein aggregates have been postulated to underlie the spectrum of clinical and pathological presentations seen across the synucleinopathies. Whereas multiple system atrophy (MSA) is associated with a...
5.
So R, Watts J
J Mol Biol
. 2023 Feb;
435(12):168011.
PMID: 36792008
The synucleinopathies, which include Parkinson's disease, dementia with Lewy bodies, and multiple system atrophy, are a class of human neurodegenerative disorders unified by the presence of α-synuclein aggregates in the...
6.
Martinez-Valbuena I, Visanji N, Kim A, Lau H, So R, Alshimemeri S, et al.
Transl Neurodegener
. 2022 Feb;
11(1):7.
PMID: 35125105
Background: Multiple system atrophy (MSA) is a neurodegenerative condition characterized by variable combinations of parkinsonism, autonomic failure, cerebellar ataxia and pyramidal features. Although the distribution of synucleinopathy correlates with the...
7.
Lau A, So R, Lau H, Sang J, Ruiz-Riquelme A, Fleck S, et al.
Nat Neurosci
. 2019 Dec;
23(1):21-31.
PMID: 31792467
The clinical and pathological differences between synucleinopathies such as Parkinson's disease and multiple system atrophy have been postulated to stem from unique strains of α-synuclein aggregates, akin to what occurs...
8.
So R, Chung S, Lau H, Watts J, Gaudette E, Al-Azzawi Z, et al.
Mol Neurodegener
. 2019 Nov;
14(1):41.
PMID: 31727120
The adoption of CRISPR-Cas9 technology for functional genetic screens has been a transformative advance. Due to its modular nature, this technology can be customized to address a myriad of questions....
9.
Liu J, Amar F, Corona C, So R, Andrews S, Nagy P, et al.
Front Mol Neurosci
. 2018 Mar;
11:62.
PMID: 29599707
Activating transcription factor 4 (ATF4) plays important physiologic roles in the brain including regulation of learning and memory as well as neuronal survival and death. Yet, outside of translational regulation...