R A I de Vos
Overview
Explore the profile of R A I de Vos including associated specialties, affiliations and a list of published articles.
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Articles
27
Citations
987
Followers
0
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Recent Articles
1.
Said S, Wester A, Brouwer R, Monnink S, Koning-Mulder W, de Vos R, et al.
Neth Heart J
. 2015 Feb;
13(6):242-243.
PMID: 25696499
No abstract available.
2.
Seidel K, Brunt E, de Vos R, Dijk F, van der Want H, Rub U, et al.
Clin Neuropathol
. 2009 Oct;
28(5):344-9.
PMID: 19788049
Neuronal protein aggregates are considered as pathological hallmarks of various human neurodegenerative diseases, including the so-called CAG-repeat disorders, such as spinocerebellar ataxia Type 6 (SCA6). Since the immunocytochemical findings of...
3.
Gierga K, Schelhaas H, Brunt E, Seidel K, Scherzed W, Egensperger R, et al.
Neuropathol Appl Neurobiol
. 2009 Feb;
35(5):515-27.
PMID: 19207264
Aims: Spinocerebellar ataxia type 6 (SCA6) is a late onset autosomal dominantly inherited ataxic disorder, which belongs to the group of CAG repeat, or polyglutamine, diseases. Although, it has long...
4.
Rub U, Brunt E, Seidel K, Gierga K, Mooy C, Kettner M, et al.
Neuropathol Appl Neurobiol
. 2007 Nov;
34(2):155-68.
PMID: 17971076
Spinocerebellar ataxia type 7 (SCA7) represents a rare and severe autosomal dominantly inherited ataxic disorder and is among the known CAG-repeat, or polyglutamine, diseases. In contrast to other currently known...
5.
Hoozemans J, van Haastert E, Eikelenboom P, de Vos R, Rozemuller J, Scheper W
Biochem Biophys Res Commun
. 2007 Jan;
354(3):707-11.
PMID: 17254549
Parkinson's disease (PD) is, at the neuropathological level, characterized by the accumulation of misfolded proteins. The presence of misfolded proteins can trigger a cellular stress response in the endoplasmic reticulum...
6.
Evert B, Schelhaas J, Fleischer H, de Vos R, Brunt E, Stenzel W, et al.
Clin Neuropathol
. 2006 Dec;
25(6):272-81.
PMID: 17140157
Objective: We analyzed the expression of the inflammatory mediators IL-1beta, IL-1ra, IL-6 and the transcription factors IRF-1 and C/EBPdelta (previously identified in a transgenic model of spinocerebellar ataxia type 3...
7.
Rub U, Brunt E, Petrasch-Parwez E, Schols L, Theegarten D, Auburger G, et al.
Neuropathol Appl Neurobiol
. 2006 Nov;
32(6):635-49.
PMID: 17083478
Dysphagia, which can lead to nutritional deficiencies, weight loss and dehydration, represents a risk factor for aspiration pneumonia. Although clinical studies have reported the occurrence of dysphagia in patients with...
8.
van Leeuwen F, Kros J, Kamphorst W, van Schravendijk C, de Vos R
Biochem Soc Trans
. 2006 Oct;
34(Pt 5):738-42.
PMID: 17052186
Neuronal homoeostasis requires a constant balance between biosynthetic and catabolic processes. Eukaryotic cells primarily use two distinct mechanisms for degradation: the proteasome and autophagy of aggregates by the lysosomes. We...
9.
Braak H, Muller C, Rub U, Ackermann H, Bratzke H, de Vos R, et al.
J Neural Transm Suppl
. 2006 Oct;
(70):89-97.
PMID: 17017514
Parkinson's disease (PD) is a multisystem disorder in which predisposed neuronal types in specific regions of the human peripheral, enteric, and central nervous systems become progressively involved. A staging procedure...
10.
van Leeuwen F, van Tijn P, Sonnemans M, Hobo B, Mann D, Van Broeckhoven C, et al.
Neurology
. 2006 Jan;
66(2 Suppl 1):S86-92.
PMID: 16432153
Frameshift (+1) proteins such as APP(+1) and UBB(+1) accumulate in sporadic cases of Alzheimer disease (AD) and in older subjects with Down syndrome (DS). We investigated whether these proteins also...