Prita R Asih
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Explore the profile of Prita R Asih including associated specialties, affiliations and a list of published articles.
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23
Citations
645
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Recent Articles
1.
Ramezani M, Fernando M, Eslick S, Asih P, Shadfar S, Bandara E, et al.
Front Neurosci
. 2023 Nov;
17:1297984.
PMID: 38033541
Alzheimer's disease (AD) is the most common form of dementia. AD is a progressive neurodegenerative disorder characterized by cognitive dysfunction, including learning and memory deficits, and behavioral changes. Neuropathology hallmarks...
2.
Prikas E, Paric E, Asih P, Stefanoska K, Stefen H, Fath T, et al.
EMBO J
. 2022 Aug;
41(18):e10242.
PMID: 35993331
Microtubule-associated protein tau is a central factor in Alzheimer's disease and other tauopathies. However, the physiological functions of tau are unclear. Here, we used proximity-labelling proteomics to chart tau interactomes...
3.
Stefanoska K, Gajwani M, Tan A, Ahel H, Asih P, Volkerling A, et al.
Sci Adv
. 2022 Jul;
8(27):eabl8809.
PMID: 35857446
Hyperphosphorylation of the neuronal tau protein is a hallmark of neurodegenerative tauopathies such as Alzheimer's disease. A central unanswered question is why tau becomes progressively hyperphosphorylated. Here, we show that...
4.
Asih P, Poljak A, Kassiou M, Ke Y, Ittner L
PLoS One
. 2022 May;
17(5):e0254296.
PMID: 35522669
The translocator protein (TSPO) has been implicated in mitochondrial transmembrane cholesterol transport, brain inflammation, and other mitochondrial functions. It is upregulated in glial cells during neuroinflammation in Alzheimer's disease. High...
5.
Asih P, Stefanoska K, Prikas E, Ittner A
Neuroscience
. 2022 Jan;
484:53-65.
PMID: 35031398
Persistent improvement of cognitive deficits in Alzheimer's disease (AD), a common form of dementia, is an unattained therapeutic objective. Gene therapy holds promise for treatment of familial and sporadic forms...
6.
Chatterjee P, Pedrini S, Ashton N, Tegg M, Goozee K, Singh A, et al.
Alzheimers Dement
. 2021 Sep;
18(6):1141-1154.
PMID: 34494715
Introduction: This study involved a parallel comparison of the diagnostic and longitudinal monitoring potential of plasma glial fibrillary acidic protein (GFAP), total tau (t-tau), phosphorylated tau (p-tau181 and p-tau231), and...
7.
Chatterjee P, Pedrini S, Stoops E, Goozee K, Villemagne V, Asih P, et al.
Transl Psychiatry
. 2021 Jan;
11(1):27.
PMID: 33431793
Glial fibrillary acidic protein (GFAP), an astrocytic cytoskeletal protein, can be measured in blood samples, and has been associated with Alzheimer's disease (AD). However, plasma GFAP has not been investigated...
8.
Asih P, Prikas E, Stefanoska K, Tan A, Ahel H, Ittner A
Front Mol Neurosci
. 2020 Oct;
13:570586.
PMID: 33013322
Mitogen-activated protein (MAP) kinases are a central component in signaling networks in a multitude of mammalian cell types. This review covers recent advances on specific functions of p38 MAP kinases...
9.
van der Hoven J, van Hummel A, Przybyla M, Asih P, Gajwani M, Feiten A, et al.
Sci Rep
. 2020 Aug;
10(1):13845.
PMID: 32796905
Antibodies have been explored extensively as a potential therapeutic for Alzheimer's disease, where amyloid-β (Aβ) peptides and the tau protein deposit in patient brains. While the major focus of antibody-based...
10.
Chatterjee P, Cheong Y, Bhatnagar A, Goozee K, Wu Y, McKay M, et al.
J Neurochem
. 2020 Jul;
159(2):389-402.
PMID: 32679614
Alzheimer's disease (AD) is a progressive neurodegenerative disorder that currently has no cure. Identifying biochemical changes associated with neurodegeneration prior to symptom onset, will provide insight into the biological mechanisms...