Peter M Visscher
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Explore the profile of Peter M Visscher including associated specialties, affiliations and a list of published articles.
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430
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66304
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Recent Articles
1.
Couvy-Duchesne B, Frouin V, Bouteloup V, Koussis N, Sidorenko J, Jiang J, et al.
Hum Brain Mapp
. 2025 Feb;
46(2):e70089.
PMID: 39907291
Alzheimer's disease (AD) brain markers are needed to select people with early-stage AD for clinical trials and as quantitative endpoint measures in trials. Using 10 clinical cohorts (N = 9140)...
2.
Visscher P, Gyngell C, Yengo L, Savulescu J
Nature
. 2025 Jan;
637(8046):637-645.
PMID: 39779842
Polygenic genome editing in human embryos and germ cells is predicted to become feasible in the next three decades. Several recent books and academic papers have outlined the ethical concerns...
3.
Davies N, Hemani G, Neiderhiser J, Martin H, Mills M, Visscher P, et al.
Nature
. 2024 Oct;
634(8035):795-803.
PMID: 39443775
Biobanks aim to improve our understanding of health and disease by collecting and analysing diverse biological and phenotypic information in large samples. So far, biobanks have largely pursued a population-based...
4.
Sidorenko J, Couvy-Duchesne B, Kemper K, Moen G, Bhatta L, Asvold B, et al.
Nat Genet
. 2024 Oct;
56(11):2352-2360.
PMID: 39375568
Linkage studies have successfully mapped loci underlying monogenic disorders, but mostly failed when applied to common diseases. Conversely, genome-wide association studies (GWASs) have identified replicable associations between thousands of SNPs...
5.
Wu Y, Zheng Z, Thibaut L, Goddard M, Wray N, Visscher P, et al.
Res Sq
. 2024 Aug;
PMID: 39149449
Fine-mapping refines genotype-phenotype association signals to identify causal variants underlying complex traits. However, current methods typically focus on individual genomic segments without considering the global genetic architecture. Here, we demonstrate...
6.
Wu Y, Zheng Z, Thibaut L, Goddard M, Wray N, Visscher P, et al.
medRxiv
. 2024 Jul;
PMID: 39072021
Fine-mapping refines genotype-phenotype association signals to identify causal variants underlying complex traits. However, current methods typically focus on individual genomic segments without considering the global genetic architecture. Here, we demonstrate...
7.
Kemper K, Sidorenko J, Wang H, Hayes B, Wray N, Yengo L, et al.
Nat Commun
. 2024 May;
15(1):3776.
PMID: 38710707
The causes of temporal fluctuations in adult traits are poorly understood. Here, we investigate the genetic determinants of within-person trait variability of 8 repeatedly measured anthropometric traits in 50,117 individuals...
8.
Keaton J, Kamali Z, Xie T, Vaez A, Williams A, Goleva S, et al.
Nat Genet
. 2024 Apr;
56(5):778-791.
PMID: 38689001
Hypertension affects more than one billion people worldwide. Here we identify 113 novel loci, reporting a total of 2,103 independent genetic signals (P < 5 × 10) from the largest...
9.
Zheng Z, Liu S, Sidorenko J, Wang Y, Lin T, Yengo L, et al.
Nat Genet
. 2024 Apr;
56(5):767-777.
PMID: 38689000
We develop a method, SBayesRC, that integrates genome-wide association study (GWAS) summary statistics with functional genomic annotations to improve polygenic prediction of complex traits. Our method is scalable to whole-genome...
10.
Hatton A, Cheng F, Lin T, Shen R, Chen J, Zheng Z, et al.
Nat Commun
. 2024 Mar;
15(1):2713.
PMID: 38548728
DNA methylation is an ideal trait to study the extent of the shared genetic control across ancestries, effectively providing hundreds of thousands of model molecular traits with large QTL effect...