Paula Lazcoz
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Explore the profile of Paula Lazcoz including associated specialties, affiliations and a list of published articles.
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10
Citations
166
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Recent Articles
1.
Blanco-Luquin I, Lazcoz P, Celay J, Castresana J, Encio I
Pharmaceuticals (Basel)
. 2021 Nov;
14(11).
PMID: 34832966
Neuroblastoma is the most frequent malignant extracranial solid tumor of infancy. The overall objective of this work consists of determining the presence of alterations in the p53/MDM2/p14ARF signaling pathway in...
2.
Celay J, Blanco I, Lazcoz P, Rotinen M, Castresana J, Encio I
PLoS One
. 2013 May;
8(5):e62771.
PMID: 23650528
To determine the effect of retinoic acid (RA) in neuroblastoma we treated RA sensitive neuroblastoma cell lines with 9-cis RA or ATRA for 9 days, or for 5 days followed...
3.
Munoz J, Inda M, Lazcoz P, Zazpe I, Fan X, Alfaro J, et al.
ISRN Neurol
. 2012 Mar;
2012:576578.
PMID: 22389839
While allelic losses and mutations of tumor suppressor genes implicated in the etiology of astrocytoma have been widely assessed, the role of epigenetics is still a matter of study. We...
4.
Schiapparelli P, Shahi M, Enguita-German M, Johnsen J, Kogner P, Lazcoz P, et al.
Cancer Lett
. 2011 Aug;
310(2):222-31.
PMID: 21803487
Sonic hedgehog (Hh) developmental pathway deregulation has been proven to play an essential role in several malignancies as neuroblastoma. We found that Hh signaling is active in neuroblastoma, as most...
5.
Schiapparelli P, Enguita-German M, Balbuena J, Rey J, Lazcoz P, Castresana J
Oncol Rep
. 2010 Sep;
24(5):1355-62.
PMID: 20878131
Neuroblastoma is the most common extracranial solid tumor in children, accounting for up to 10% of all childhood malignancies. Cellular heterogeneity is a hallmark of this embryonal cancer, as distinct...
6.
Lorente A, Mueller W, Urdangarin E, Lazcoz P, Lass U, von Deimling A, et al.
Brain Pathol
. 2008 Jul;
19(2):279-92.
PMID: 18616639
Methylation of CpG islands in gene promoters can lead to gene silencing. Together with deletion or mutation, it may cause a loss of function of tumor suppressor genes. RASSF1A (3p21.3),...
7.
Lorente A, Mueller W, Urdangarin E, Lazcoz P, von Deimling A, Castresana J
BMC Cancer
. 2008 Feb;
8:61.
PMID: 18298842
Background: We present two melting curve analysis (MCA)-based semiquantitative real time PCR techniques to detect the promoter methylation status of genes. The first, MCA-MSP, follows the same principle as standard...
8.
Lazcoz P, Munoz J, Nistal M, Pestana A, Encio I, Castresana J
Cancer Genet Cytogenet
. 2007 Mar;
174(1):1-8.
PMID: 17350460
Tumor suppressor genes can be inactivated by various mechanisms, including promoter hypermethylation and loss of heterozygosity. We screened the 10q locus for loss of heterozygosity and the promoter methylation status...
9.
Lazcoz P, Munoz J, Nistal M, Pestana A, Encio I, Castresana J
BMC Cancer
. 2006 Oct;
6:254.
PMID: 17064406
Background: Epigenetic alterations and loss of heterozygosity are mechanisms of tumor suppressor gene inactivation. A new carcinogenic pathway, targeting the RAS effectors has recently been documented. RASSF1A, on 3p21.3, and...
10.
Munoz J, Lazcoz P, Inda M, Nistal M, Pestana A, Encio I, et al.
Int J Cancer
. 2004 Mar;
109(5):673-9.
PMID: 14999773
Neuroblastoma is the most common pediatric solid tumor. Although many allelic imbalances have been described, a bona fide tumor suppressor gene for this disease has not been found yet. In...