Paul G Weightman Potter
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Explore the profile of Paul G Weightman Potter including associated specialties, affiliations and a list of published articles.
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9
Citations
75
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Recent Articles
1.
Marina C, de Castro R, Bellozi P, Cruz A, Burgel P, Potter P, et al.
Infect Immun
. 2024 Dec;
93(2):e0048724.
PMID: 39714095
Dormancy is an adaptation in which cells reduce their metabolism, transcription, and translation to stay alive under stressful conditions, preserving the capacity to reactivate once the environment reverts to favorable...
2.
Firth W, Pye K, Potter P
Clin Sci (Lond)
. 2024 Apr;
138(8):515-536.
PMID: 38652065
Once considered passive cells of the central nervous system (CNS), glia are now known to actively maintain the CNS parenchyma; in recent years, the evidence for glial functions in CNS...
3.
Potter P, Ellacott K, Randall A, Beall C
Cells
. 2022 Nov;
11(21).
PMID: 36359822
Astrocytes contribute to glutamatergic signalling, which is required for hypoglycaemia counterregulation and is impaired by recurrent insulin-induced hypoglycaemia. This study examined the glutamate response of astrocytes when challenged with acute...
4.
Cruz A, Partridge K, Malekizadeh Y, Vlachaki Walker J, Potter P, Pye K, et al.
Front Endocrinol (Lausanne)
. 2022 Jan;
12:697445.
PMID: 34975743
Aim: We evaluated the efficacy of a novel brain permeable "metformin-like" AMP-activated protein kinase activator, R481, in regulating glucose homeostasis. Materials And Methods: We used glucose sensing hypothalamic GT1-7 neuronal...
5.
Potter P, Washer S, Jeffries A, Holley J, Gutowski N, Dempster E, et al.
Front Endocrinol (Lausanne)
. 2021 Jun;
12:671724.
PMID: 34122346
Aims/hypothesis: Recurrent hypoglycaemia (RH) is a major side-effect of intensive insulin therapy for people with diabetes. Changes in hypoglycaemia sensing by the brain contribute to the development of impaired counterregulatory...
6.
Robb J, Hammad N, Potter P, Chilton J, Beall C, Ellacott K
Glia
. 2020 Apr;
68(11):2246-2263.
PMID: 32277522
Inflammation and metabolism are intrinsically linked with inflammatory stimuli inducing metabolic changes in cells and, in turn, metabolic capacity determining cellular inflammatory responses. Although well characterized in peripheral immune cells...
7.
Robb J, Morrissey N, Potter P, Smithers H, Beall C, Ellacott K
Neuroscience
. 2019 Nov;
447:167-181.
PMID: 31765625
Chronic low-grade inflammation is a feature of the pathophysiology of obesity and diabetes in the CNS as well as peripheral tissues. Glial cells are critical mediators of the response to...
8.
Potter P, Vlachaki Walker J, Robb J, Chilton J, Williamson R, Randall A, et al.
Diabetologia
. 2018 Oct;
62(1):187-198.
PMID: 30293112
Aims/hypothesis: Hypoglycaemia is a major barrier to good glucose control in type 1 diabetes. Frequent hypoglycaemic episodes impair awareness of subsequent hypoglycaemic bouts. Neural changes underpinning awareness of hypoglycaemia are...
9.
Vlachaki Walker J, Robb J, Cruz A, Malhi A, Potter P, Ashford M, et al.
Diabetes Obes Metab
. 2017 Feb;
19(7):997-1005.
PMID: 28211632
Aim: To test the hypothesis that, given the role of AMP-activated protein kinase (AMPK) in regulating intracellular ATP levels, AMPK may alter ATP release from astrocytes, the main sources of...