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P T Francis

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Articles 96
Citations 1666
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Recent Articles
1.
Clarke E, Jantrachotechatchawan C, Buhidma Y, Broadstock M, Yu L, Howlett D, et al.
Neurochem Int . 2019 Jul; 129:104502. PMID: 31299418
Heterozygous mutations in GBA1, the gene which encodes the lysosomal enzyme glucocerebrosidase (GCase), are a strong genetic risk factor for the development of Lewy body dementia (LBD). Until this point...
2.
Patel T, Brookes K, Turton J, Chaudhury S, Guetta-Baranes T, Guerreiro R, et al.
Neuropathol Appl Neurobiol . 2017 Nov; 44(5):506-521. PMID: 29181857
Aim: Late-onset Alzheimer's disease (LOAD) accounts for 95% of all Alzheimer's cases and is genetically complex in nature. Overlapping clinical and neuropathological features between AD, FTD and Parkinson's disease highlight...
3.
Cidis Meltzer C, Francis P
Dialogues Clin Neurosci . 2011 Oct; 3(3):167-80. PMID: 22034395
Remarkable and continued growth in the field of brain aging research has been fueled by a confluence of factors. Developments in molecular biology, imaging, and genetics coupled with the imperative...
4.
Kirvell S, Elliott M, Kalaria R, Hortobagyi T, Ballard C, Francis P
Neurology . 2010 Nov; 75(20):1803-9. PMID: 21079182
Objectives: Vascular dementia (VaD) accounts for approximately 15%-20% of all dementias, but the relationship of progressive cognitive impairment to neurochemical changes is poorly understood. We have therefore investigated glutamatergic synaptic...
5.
Francis P
Curr Mol Pharmacol . 2009 Dec; 2(1):77-82. PMID: 20021448
Behavioural symptoms are a significant problem in Alzheimer's disease (AD). Symptoms including agitation/aggression and psychosis reduce patient quality of life, significantly increase caregiver burden, and often trigger nursing home placement....
6.
Tsang S, Francis P, Esiri M, Wong P, Chen C, Lai M
Psychopharmacology (Berl) . 2008 Apr; 198(2):251-9. PMID: 18373228
Rationale: Neuropsychiatric behaviours in Alzheimer's disease (AD) patients have been associated with neocortical alterations of presynaptic cholinergic and muscarinic M2 receptor markers. In contrast, it is unclear whether non-M2 muscarinic...
7.
Bowen D, Procter A, Mann D, Snowden J, Esiri M, Neary D, et al.
Psychopharmacology (Berl) . 2007 Nov; 196(4):603-10. PMID: 18026720
Rationale: Information is sparse on neurotransmitter deficiencies in frontotemporal dementia (FTD), in particular with reference to distinct histological subgroups and Alzheimer's disease (AD). Objectives: To evaluate in FTD with the...
8.
Holmes C, McCulley M, Nicoll J, Alder J, Chen C, Francis P
Neurosci Lett . 2007 May; 420(1):58-60. PMID: 17481814
A common intronic single nucleotide polymorphism (T102C) in the 5-HT2A receptor gene is associated with the development of different neuropsychiatric symptoms, including hallucinations and depressive symptoms in Alzheimer's disease (AD)....
9.
Jacobs E, Williams R, Francis P
Neuroscience . 2006 Jan; 138(2):511-22. PMID: 16413130
Besides formation of neurofibrillary tangles and neuron loss, the Alzheimer's disease brain is characterized by neuritic plaques consisting of beta-amyloid peptide deposits and impaired neurotransmission. The proteins Munc18a, Munc18-interacting protein...
10.
Garcia-Alloza M, Gil-Bea F, Diez-Ariza M, Chen C, Francis P, Lasheras B, et al.
Neuropsychologia . 2005 Feb; 43(3):442-9. PMID: 15707619
Neuropsychiatric symptoms seen in Alzheimer's disease (AD) are not simply a consequence of neurodegeneration, but probably result from differential neurotransmitter alterations, which some patients are more at risk of than...