Olga Gielen
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Explore the profile of Olga Gielen including associated specialties, affiliations and a list of published articles.
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26
Citations
512
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Recent Articles
1.
Aertgeerts M, Meyers S, Gielen O, Lamote J, Dewaele B, Tajdar M, et al.
Hemasphere
. 2025 Feb;
9(2):e70085.
PMID: 39944233
High hyperdiploid (HeH) B-cell acute lymphoblastic leukemia (B-ALL) is the most prevalent subtype of childhood ALL. This leukemia is characterized by trisomies and tetrasomies of specific chromosomes and additional point...
2.
Lauwereins L, Van Thillo Q, Demeyer S, Mentens N, Provost S, Jacobs K, et al.
Leukemia
. 2025 Jan;
39(3):568-576.
PMID: 39838044
T-cell acute lymphoblastic leukemia (T-ALL) is an aggressive hematological disease originating from the malignant transformation of T-cell progenitors, caused by the accumulation of genetic aberrations. One-fifth of T-ALL patients are...
3.
Meyers S, Gielen O, Cools J, Demeyer S
Haematologica
. 2024 May;
109(10):3167-3181.
PMID: 38813729
T-cell acute lymphoblastic leukemia (T-ALL) is an aggressive type of leukemia caused by accumulation of multiple genetic alterations in T-cell progenitors. However, for many genes it remains unknown how their...
4.
Vandersmissen C, Prieto C, Gielen O, Jacobs K, Nittner D, Maertens J, et al.
Haematologica
. 2023 Jan;
108(9):2507-2512.
PMID: 36700404
No abstract available.
5.
Vanden Bempt M, Debackere K, Demeyer S, Van Thillo Q, Meeuws N, Prieto C, et al.
Blood
. 2022 Aug;
140(23):2463-2476.
PMID: 35960849
Peripheral T-cell lymphoma (PTCL) is a heterogeneous group of hematological cancers arising from the malignant transformation of mature T cells. In a cohort of 28 PTCL cases, we identified recurrent...
6.
Thielemans N, Demeyer S, Mentens N, Gielen O, Provost S, Cools J
Haematologica
. 2022 Mar;
107(10):2304-2317.
PMID: 35354248
TAL1 is ectopically expressed in about 30% of T-cell acute lymphoblastic leukemia (T-ALL) due to chromosomal rearrangements leading to the STIL-TAL1 fusion genes or due to non-coding mutations leading to...
7.
Meyers S, Alberti-Servera L, Gielen O, Erard M, Swings T, De Bie J, et al.
Hemasphere
. 2022 Mar;
6(4):e700.
PMID: 35291210
Acute lymphoblastic leukemia (ALL) is characterized by the presence of chromosomal changes, including numerical changes, translocations, and deletions, which are often associated with additional single-nucleotide mutations. In this study, we...
8.
Rack K, De Bie J, Ameye G, Gielen O, Demeyer S, Cools J, et al.
Am J Hematol
. 2022 Feb;
97(5):548-561.
PMID: 35119131
Acute lymphoblastic leukemia (ALL) is a malignancy that can be subdivided into distinct entities based on clinical, immunophenotypic and genomic features, including mutations, structural variants (SVs), and copy number alterations...
9.
Van Thillo Q, De Bie J, Seneviratne J, Demeyer S, Omari S, Balachandran A, et al.
Nat Commun
. 2021 Jul;
12(1):4164.
PMID: 34230493
Spi-1 Proto-Oncogene (SPI1) fusion genes are recurrently found in T-cell acute lymphoblastic leukemia (T-ALL) cases but are insufficient to drive leukemogenesis. Here we show that SPI1 fusions in combination with...
10.
Govaerts I, Prieto C, Vandersmissen C, Gielen O, Jacobs K, Provost S, et al.
J Hematol Oncol
. 2021 Jun;
14(1):97.
PMID: 34167562
Background: T cell acute lymphoblastic leukemia (T-ALL) is a high-risk subtype that comprises 10-15% of childhood and 20-25% of adult ALL cases. Over 70% of T-ALL patients harbor activating mutations...