Nozomi Hayashiji
Overview
Explore the profile of Nozomi Hayashiji including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
9
Citations
173
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Hayashiji N, Kawahara G, Xu X, Fukuda T, Kerever A, Gu J, et al.
Cells
. 2023 Jan;
12(1).
PMID: 36611938
Glycosylation is an important mechanism regulating various biological processes, including intercellular signaling and adhesion. α-1,6-fucosyltransferase (Fut8) belongs to a family of enzymes that determine the terminal structure of glycans. Fut8...
2.
Shimojima M, Yuasa S, Motoda C, Yozu G, Nagai T, Ito S, et al.
Sci Rep
. 2017 Mar;
7:44312.
PMID: 28290476
Alteration of the nuclear Ca transient is an early event in cardiac remodeling. Regulation of the nuclear Ca transient is partly independent of the cytosolic Ca transient in cardiomyocytes. One...
3.
Okata S, Yuasa S, Suzuki T, Ito S, Makita N, Yoshida T, et al.
Sci Rep
. 2016 Sep;
6:34198.
PMID: 27677334
SCN5A is abundant in heart and has a major role in I. Loss-of-function mutation in SCN5A results in Brugada syndrome (BrS), which causes sudden death in adults. It remains unclear...
4.
Hashimoto H, Yuasa S, Tabata H, Tohyama S, Seki T, Egashira T, et al.
Biochem Biophys Res Commun
. 2015 Jul;
464(4):1000-1007.
PMID: 26168730
The precise assemblage of several types of cardiac precursors controls heart organogenesis. The cardiac precursors show dynamic movement during early development and then form the complicated heart structure. However, cardiomyocyte...
5.
Hayashiji N, Yuasa S, Miyagoe-Suzuki Y, Hara M, Ito N, Hashimoto H, et al.
Nat Commun
. 2015 Jun;
6:7295.
PMID: 26084207
No abstract available.
6.
Hayashiji N, Yuasa S, Miyagoe-Suzuki Y, Hara M, Ito N, Hashimoto H, et al.
Nat Commun
. 2015 Apr;
6:6745.
PMID: 25865621
Duchenne muscular dystrophy (DMD) is a chronic and life-threatening disease that is initially supported by muscle regeneration but eventually shows satellite cell exhaustion and muscular dysfunction. The life-long maintenance of...
7.
Kodaira M, Hatakeyama H, Yuasa S, Seki T, Egashira T, Tohyama S, et al.
FEBS Open Bio
. 2015 Apr;
5:219-25.
PMID: 25853038
Mitochondrial diseases are heterogeneous disorders, caused by mitochondrial dysfunction. Mitochondria are not regulated solely by nuclear genomic DNA but by mitochondrial DNA. It is difficult to develop effective therapies for...
8.
Hashimoto H, Yuasa S, Tabata H, Tohyama S, Hayashiji N, Hattori F, et al.
J Mol Cell Cardiol
. 2014 Apr;
72:241-9.
PMID: 24704900
Mammalian cardiomyocytes withdraw from the cell cycle shortly after birth, although it remains unclear how cardiomyocyte cell cycles behave during development. Compared to conventional immunohistochemistry in static observation, time-lapse imaging...
9.
Hara M, Yuasa S, Shimoji K, Onizuka T, Hayashiji N, Ohno Y, et al.
J Exp Med
. 2011 Mar;
208(4):715-27.
PMID: 21422169
After skeletal muscle injury, neutrophils, monocytes, and macrophages infiltrate the damaged area; this is followed by rapid proliferation of myoblasts derived from muscle stem cells (also called satellite cells). Although...