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Norbert Voelkel

Explore the profile of Norbert Voelkel including associated specialties, affiliations and a list of published articles. Areas
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Articles 17
Citations 472
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Recent Articles
1.
Miwa H, Sakao S, Sanada T, Suzuki H, Hata A, Shiina Y, et al.
Can J Cardiol . 2021 Feb; 37(6):913-923. PMID: 33609715
Background: The mechanism of vascular remodelling in pulmonary arterial hypertension (PAH) remains unclear. Hence, defining the origin of cells constituting intractable vascular lesions in PAH is expected to facilitate therapeutic...
2.
Spiekerkoetter E, Goncharova E, Guignabert C, Stenmark K, Kwapiszewska G, Rabinovitch M, et al.
Pulm Circ . 2019 Dec; 9(4):2045894019889775. PMID: 31798835
In order to intervene appropriately and develop disease-modifying therapeutics for pulmonary arterial hypertension, it is crucial to understand the mechanisms of disease pathogenesis and progression. We herein discuss four topics...
3.
Kovacs G, Agusti A, Barbera J, Celli B, Criner G, Humbert M, et al.
Am J Respir Crit Care Med . 2018 May; 198(8):1000-1011. PMID: 29746142
No abstract available.
4.
Shojaee S, Shojaee S, Voelkel N, Farkas L, de Wit M, Lee H
J Bronchology Interv Pulmonol . 2013 Oct; 20(4):304-8. PMID: 24162112
Background: Transforming growth factor-β1 (TGF-β1) plays a major role in pleural fibrosis and chemical pleurodesis. Approximately 50% of patients with tunneled pleural catheters (TPCs) for malignant pleural effusions (MPEs) will...
5.
Toldo S, Breckenridge D, Mezzaroma E, Van Tassell B, Shryock J, Kannan H, et al.
J Am Heart Assoc . 2013 Jan; 1(5):e002360. PMID: 23316291
Background: Despite the clear advantages of reperfusion in acute myocardial infarction, part of the myocardium is injured during reperfusion by reactive oxygen species. Reactive oxygen species activate apoptosis signal-regulating kinase-1,...
6.
Morjaria S, Grinnan D, Voelkel N
Pulm Circ . 2012 Jul; 2(2):256-7. PMID: 22837867
Congenital pulmonary valve stenosis has been associated with the development of massive pulmonary arterial (PA) dilatation. Over time, this dilatation may distort surrounding structures and lead to compression of the...
7.
Mezzaroma E, Di X, Graves P, Toldo S, Van Tassell B, Kannan H, et al.
Int J Cardiol . 2012 Feb; 156(2):231-3. PMID: 22340985
No abstract available.
8.
Toldo S, Boccellino M, Rinaldi B, Seropian I, Mezzaroma E, Severino A, et al.
Mol Med . 2011 Jun; 17(9-10):1012-21. PMID: 21637911
Diabetes is associated with an increased risk of heart failure, in part explained by endoplasmic reticulum stress and apoptosis. Protein disulfide isomerase (PDI) prevents stressed cardiomyocytes apoptosis. We hypothesized that...
9.
Toldo S, Seropian I, Mezzaroma E, Van Tassell B, Salloum F, Lewis E, et al.
J Mol Cell Cardiol . 2011 May; 51(2):244-51. PMID: 21600901
Alpha-1-antitrypsin (AAT) possesses anti-inflammatory and tissue-protective properties. Here, we studied the effects of exogenously administered AAT on caspase-1 activity and on the outcome of ischemia-reperfusion injury (I/R) in a mouse...
10.
Erzurum S, Rounds S, Stevens T, Aldred M, Aliotta J, Archer S, et al.
Am J Respir Crit Care Med . 2010 Sep; 182(12):1554-62. PMID: 20833821
The Division of Lung Diseases of the National Heart, Lung, and Blood Institute, with the Office of Rare Diseases Research, held a workshop to identify priority areas and strategic goals...