Mathis Neuhaus
Overview
Explore the profile of Mathis Neuhaus including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
4
Citations
5
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Fryklund C, Neuhaus M, Stenkula K
Am J Physiol Endocrinol Metab
. 2023 Oct;
325(6):E682-E687.
PMID: 37877798
Adipocyte dysfunction is a hallmark of systemic insulin resistance. Insulin-responsive glucose transporter 4 (GLUT4) is downregulated in the insulin resistant state, and cellular insulin responsiveness varies depending on fat depot...
2.
Neuhaus M, Fryklund C, Taylor H, Borreguero-Munoz A, Kopietz F, Ardalani H, et al.
Mol Biol Cell
. 2023 Sep;
34(12):ar124.
PMID: 37703099
Adipocyte dysfunction is a crucial driver of insulin resistance and type 2 diabetes. We identified EH domain-containing protein 2 (EHD2) as one of the most highly upregulated genes at the...
3.
Fryklund C, Neuhaus M, Moren B, Borreguero-Munoz A, Lundmark R, Stenkula K
Front Cell Dev Biol
. 2022 Sep;
10:942374.
PMID: 36158197
To accommodate surplus energy, the adipose tissue expands by increasing adipocyte size (hypertrophy) and number (hyperplasia). The presence of hypertrophic adipocytes is a key characteristic of adipose tissue dysfunction. High-fat...
4.
Fryklund C, Moren B, Neuhaus M, Periwal V, Stenkula K
Life Sci
. 2022 Apr;
299:120537.
PMID: 35398016
Aims: To accommodate surplus energy, adipose tissue expands by increasing both adipose cell size (hypertrophy) and cell number (hyperplasia). Enlarged, hypertrophic adipocytes are known to have reduced insulin response and...