Marzia Tartari
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Explore the profile of Marzia Tartari including associated specialties, affiliations and a list of published articles.
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18
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1420
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Recent Articles
1.
Lo Sardo V, Zuccato C, Gaudenzi G, Vitali B, Ramos C, Tartari M, et al.
Nat Neurosci
. 2012 Apr;
15(5):713-21.
PMID: 22466506
The Huntington's disease gene product, huntingtin, is indispensable for neural tube formation, but its role is obscure. We studied neurulation in htt-null embryonic stem cells and htt-morpholino zebrafish embryos and...
2.
Lim D, Fedrizzi L, Tartari M, Zuccato C, Cattaneo E, Brini M, et al.
J Biol Chem
. 2007 Dec;
283(9):5780-9.
PMID: 18156184
Dysfunctions of Ca2+ homeostasis and of mitochondria have been studied in immortalized striatal cells from a commonly used Huntington disease mouse model. Transcriptional changes in the components of the phosphatidylinositol...
3.
Zuccato C, Marullo M, Conforti P, MacDonald M, Tartari M, Cattaneo E
Brain Pathol
. 2007 Dec;
18(2):225-38.
PMID: 18093249
One cardinal feature of Huntington's disease (HD) is the degeneration of striatal neurons, whose survival greatly depends on the binding of cortical brain-derived neurotrophic factor (BDNF) with high-affinity (TrkB) and...
4.
Tartari M, Gissi C, Lo Sardo V, Zuccato C, Picardi E, Pesole G, et al.
Mol Biol Evol
. 2007 Dec;
25(2):330-8.
PMID: 18048403
Huntingtin is a completely soluble 3,144 amino acid (aa) protein characterized by the presence of an amino-terminal polymorphic polyglutamine (polyQ) tract, whose aberrant expansion causes the progressively neurodegenerative Huntington's disease...
5.
Candiani S, Pestarino M, Cattaneo E, Tartari M
BMC Dev Biol
. 2007 Nov;
7:127.
PMID: 18005438
Background: Huntington's disease is an inherited neurodegenerative disorder that is caused by the expansion of an N-terminal polyQ stretch in the huntingtin protein. In order to investigate the hypothesis that...
6.
Zuccato C, Belyaev N, Conforti P, Ooi L, Tartari M, Papadimou E, et al.
J Neurosci
. 2007 Jun;
27(26):6972-83.
PMID: 17596446
Huntingtin is a protein that is mutated in Huntington's disease (HD), a dominant inherited neurodegenerative disorder. We previously proposed that, in addition to the gained toxic activity of the mutant...
7.
Rigamonti D, Bolognini D, Mutti C, Zuccato C, Tartari M, Sola F, et al.
J Biol Chem
. 2007 Jun;
282(34):24554-62.
PMID: 17565993
Increased levels of the repressor element 1/neuron restrictive silencer element (RE1/NRSE) silencing activity promoter, and a consequent reduction in the transcription of many RE1/NRSE-bearing neuronal genes, including brain-derived neurotrophic factor...
8.
Gissi C, Pesole G, Cattaneo E, Tartari M
BMC Genomics
. 2006 Nov;
7:288.
PMID: 17092333
Background: To gain insight into the evolutionary features of the huntingtin (htt) gene in Chordata, we have sequenced and characterized the full-length htt mRNA in the ascidian Ciona intestinalis, a...
9.
Di Maria E, Marasco A, Tartari M, Ciotti P, Abbruzzese G, Novelli G, et al.
Neurobiol Dis
. 2006 Aug;
24(2):274-9.
PMID: 16905325
Huntington's disease (HD) is a late-onset, autosomal dominant neurodegenerative disease caused by a CAG trinucleotide expansion. The number of repeats on the HD chromosome explains most of the variability in...
10.
Cattaneo E, Zuccato C, Tartari M
Nat Rev Neurosci
. 2005 Nov;
6(12):919-30.
PMID: 16288298
Several neurological diseases are characterized by the altered activity of one or a few ubiquitously expressed cell proteins, but it is not known how these normal proteins turn into harmful...