Martin Ungerer
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Explore the profile of Martin Ungerer including associated specialties, affiliations and a list of published articles.
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Recent Articles
11.
Diana T, Holthoff H, Fassbender J, Wuster C, Kanitz M, Kahaly G, et al.
Eur Thyroid J
. 2021 Jan;
9(Suppl 1):51-58.
PMID: 33511085
Introduction: A novel long-term murine model for Graves' disease (GD) using repeated, long-term immunizations with recombinant adenovirus expressing the extracellular A-subunit of the human thyrotropin receptor (Ad-TSHR) was applied to...
12.
Degen H, Gavvovidis I, Blankenstein T, Uhland K, Ungerer M
Thyroid
. 2020 Nov;
31(6):950-963.
PMID: 33208049
Antigen-specific lymphocytes are increasingly investigated in autoimmune diseases and immune therapies. We sought to identify thyrotropin receptor (TSHR)-specific lymphocytes in mouse models of Graves' disease, including Graves' patient-specific immunotype human...
13.
Li Z, Goebel S, Reimann A, Ungerer M
Sci Rep
. 2020 Nov;
10(1):19849.
PMID: 33199754
A novel Histo-ELISA technique is intended to facilitate quantification of target tissue proteins in a tissue section and involves the selection of target regions in the tissue section, application of...
14.
Holthoff H, Uhland K, Kovacs G, Reimann A, Adler K, Wenhart C, et al.
J Endocrinol
. 2020 Jun;
246(2):135-147.
PMID: 32573180
Graves' disease is an autoimmune disorder, which is characterized by stimulatory antibodies targeting the human thyrotropin receptor (TSHR), resulting in hyperthyroidism and multiple organ damage. We systematically investigated monomeric and...
15.
Zhang D, Ebrahim M, Adler K, Blanchet X, Jamasbi J, Megens R, et al.
Thromb Haemost
. 2020 Jun;
120(6):977-993.
PMID: 32492725
Glycoprotein VI (GPVI), a platelet collagen receptor, is crucial in mediating atherothrombosis. Besides collagen, injured plaques expose tissue factor (TF) that triggers fibrin formation. Previous studies reported that GPVI also...
16.
Zeibig S, Buttcher M, Goebel S, Pauli J, Hunger A, Ungerer M, et al.
Cell Physiol Biochem
. 2019 Mar;
52(4):681-695.
PMID: 30921507
Background/aims: Oxidative modifications of low-density lipoprotein (ox-LDL) play a key role in initial steps of atheroprogression possibly via specific scavenger receptors on inflammatory and endothelial cells. Amongst others, CD68 might...
17.
Fassbender J, Holthoff H, Li Z, Ungerer M
Thyroid
. 2019 Jan;
29(2):258-267.
PMID: 30618332
Background: Cyclic peptides derived from some cylindrical loops of the leucine-rich repeat domain (LRD) of the thyrotropin receptor (TSHR) have been shown to treat disease manifestations in a mouse model...
18.
Boivin-Jahns V, Uhland K, Holthoff H, Beyersdorf N, Kocoski V, Kerkau T, et al.
PLoS One
. 2018 Aug;
13(8):e0201160.
PMID: 30125285
Rationale: Despite advances in pharmacotherapy, heart failure still incurs significant morbidity and mortality. Stimulating antibodies directed against the secondextracellular loop of the human ß1-adrenergic receptor (anti-ß1EC2) cause myocyte damage and...
19.
Ungerer M, Fabbender J, Holthoff H
Front Biosci (Landmark Ed)
. 2018 May;
23(11):2044-2052.
PMID: 29772544
Graves´ disease is an autoimmune disorder, which is characterized by stimulatory antibodies targeting the human thyrotropin receptor (TSHR), resulting in hyperthyroidism and multiple organ damage. The disease can be modelled...
20.
Ebrahim M, Jamasbi J, Adler K, Megens R, MBengue Y, Blanchet X, et al.
Thromb Haemost
. 2018 Jan;
118(2):351-361.
PMID: 29378359
Platelet glycoprotein VI (GPVI) acts as a decisive collagen receptor in atherothrombosis. Besides collagen, injured atherosclerotic plaques expose tissue factor (TF) that triggers fibrin formation. Two recent studies reported that...