Markus S Huber-Lang
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Explore the profile of Markus S Huber-Lang including associated specialties, affiliations and a list of published articles.
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40
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2240
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Recent Articles
1.
Mosenlechner M, Schlosser D, Braumuller S, Dorfer L, Mannes M, Kawach R, et al.
Shock
. 2024 Dec;
63(3):448-455.
PMID: 39637172
Background: Sepsis continues to pose a significant threat to human life and represents a substantial financial burden. In addition to replicative stress resulting from telomeric loss, recent studies have identified...
2.
Stahel P, Weckbach S, Huber-Lang M, Stahel V, Barnum S
Front Surg
. 2023 Feb;
10:1132752.
PMID: 36793320
No abstract available.
3.
Senescent cells suppress macrophage-mediated corpse removal via upregulation of the CD47-QPCT/L axis
Schloesser D, Lindenthal L, Sauer J, Chung K, Chavakis T, Griesser E, et al.
J Cell Biol
. 2022 Dec;
222(2).
PMID: 36459066
Progressive accrual of senescent cells in aging and chronic diseases is associated with detrimental effects in tissue homeostasis. We found that senescent fibroblasts and epithelia were not only refractory to...
4.
Karasu E, Demmelmaier J, Kellermann S, Holzmann K, Kohl J, Schmidt C, et al.
Front Immunol
. 2020 Sep;
11:1789.
PMID: 32983087
Initially underestimated as platelet dust, extracellular vesicles are continuously gaining interest in the field of inflammation. Various studies addressing inflammatory diseases have shown that microvesicles (MVs) originating from different cell...
5.
Huber-Lang M, Ignatius A, Kohl J, Mannes M, Braun C
Br J Pharmacol
. 2020 Sep;
178(14):2863-2879.
PMID: 32880897
Physical trauma represents a major global burden. The trauma-induced response, including activation of the innate immune system, strives for regeneration but can also lead to post-traumatic complications. The complement cascade...
6.
Halbgebauer R, Kellermann S, Schafer F, Weckbach S, Weiss M, Barth E, et al.
Scand J Immunol
. 2019 Oct;
91(2):e12837.
PMID: 31622512
After severe trauma, the resulting excessive inflammatory response is countered by compensatory anti-inflammatory mechanisms. The systemic inflammatory response to trauma enhanced by inappropriately timed surgical second hits may be detrimental...
7.
Messerer D, Denk S, Fohr K, Halbgebauer R, Braun C, Hones F, et al.
Mediators Inflamm
. 2018 Jul;
2018:2052356.
PMID: 30002598
Background: Polymorphonuclear granulocytes (PMN) play a crucial role in host defense. Physiologically, exposure of PMN to the complement activation product C5a results in a protective response against pathogens, whereas in...
8.
Braun C, Kalbitz M, Halbgebauer R, Eisele P, Messerer D, Weckbach S, et al.
PLoS One
. 2017 Oct;
12(10):e0187327.
PMID: 29084268
Evidence is emerging that systemic inflammation after trauma drives structural and functional impairment of cardiomyocytes and leads to cardiac dysfunction, thus worsening the outcome of polytrauma patients. This study investigates...
9.
Fattahi F, Kalbitz M, Malan E, Abe E, Jajou L, Huber-Lang M, et al.
FASEB J
. 2017 Jun;
31(9):4129-4139.
PMID: 28572445
Polymicrobial sepsis in mice causes myocardial dysfunction after generation of the complement anaphylatoxin, complement component 5a (C5a). C5a interacts with its receptors on cardiomyocytes (CMs), resulting in redox imbalance and...
10.
Denk S, Neher M, Messerer D, Wiegner R, Nilsson B, Rittirsch D, et al.
J Immunol
. 2017 May;
198(12):4846-4854.
PMID: 28490576
During sepsis, excessive activation of the complement system with generation of the anaphylatoxin C5a results in profound disturbances in crucial neutrophil functions. Moreover, because neutrophil activity is highly dependent on...