Mark J Nijland
Overview
Explore the profile of Mark J Nijland including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
42
Citations
1077
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Cox L, Puppala S, Chan J, Riojas A, Lange K, Birnbaum S, et al.
J Dev Orig Health Dis
. 2024 Nov;
15:e27.
PMID: 39506415
Previous studies in rodents suggest that mismatch between fetal and postnatal nutrition predisposes individuals to metabolic diseases. We hypothesized that in nonhuman primates (NHP), fetal programming of maternal undernutrition (MUN)...
2.
Pereira S, Diniz M, Tavares L, Cunha-Oliveira T, Li C, Cox L, et al.
Int J Mol Sci
. 2023 Oct;
24(20).
PMID: 37894873
Intra-uterine growth restriction (IUGR) is a common cause of fetal/neonatal morbidity and mortality and is associated with increased offspring predisposition for cardiovascular disease (CVD) development. Mitochondria are essential organelles in...
3.
Bishop A, Spradling-Reeves K, Shade R, Lange K, Birnbaum S, Favela K, et al.
J Med Primatol
. 2022 Jul;
51(6):329-344.
PMID: 35855511
Background: Poor nutrition during fetal development programs postnatal kidney function. Understanding postnatal consequences in nonhuman primates (NHP) is important for translation to our understanding the impact on human kidney function...
4.
Pereira S, Tavares L, Duarte A, Baldeiras I, Cunha-Oliveira T, Martins J, et al.
Clin Sci (Lond)
. 2021 Apr;
135(9):1103-1126.
PMID: 33899910
Poor maternal nutrition in pregnancy affects fetal development, predisposing offspring to cardiometabolic diseases. The role of mitochondria during fetal development on later-life cardiac dysfunction caused by maternal nutrient reduction (MNR)...
5.
Light L, Bartlett T, Poyas A, Nijland M, Huber H, Li C, et al.
J Med Primatol
. 2018 May;
PMID: 29749628
Background: We hypothesized that maternal nutrient restriction (NR) would increase activity and behavioral indicators of anxiety (self-directed behaviors, SDBs) in captive baboons (Papio sp.) and result in more protective maternal...
6.
Spradling-Reeves K, Glenn J, Lange K, Kuhn N, Coalson J, Nijland M, et al.
J Med Primatol
. 2018 Apr;
47(3):157-171.
PMID: 29603257
Background: Little is known about the repertoire of non-human primate kidney genes expressed throughout development. The present work establishes an understanding of the primate renal transcriptome during fetal development in...
7.
Rakers F, Bischoff S, Schiffner R, Haase M, Rupprecht S, Kiehntopf M, et al.
Am J Obstet Gynecol
. 2015 Jul;
213(5):684.e1-9.
PMID: 26212181
Objective: We sought to evaluate whether in addition to cortisol, catecholamines also transfer psychosocial stress indirectly to the fetus by decreasing uterine blood flow (UBF) and increasing fetal anaerobic metabolism...
8.
Jimenez-Chillaron J, Nijland M, Ascensao A, Sardao V, Magalhaes J, Hitchler M, et al.
Epigenetics
. 2015 Mar;
10(4):259-73.
PMID: 25774863
Epigenetics, or regulation of gene expression independent of DNA sequence, is the missing link between genotype and phenotype. Epigenetic memory, mediated by histone and DNA modifications, is controlled by a...
9.
Pereira S, Oliveira P, Tavares L, Moreno A, Cox L, Nathanielsz P, et al.
Am J Physiol Renal Physiol
. 2015 Mar;
308(11):F1217-28.
PMID: 25761880
Early life malnutrition results in structural alterations in the kidney, predisposing offspring to later life renal dysfunction. Kidneys of adults who were growth restricted at birth have substantial variations in...
10.
Kavitha J, Rosario F, Nijland M, McDonald T, Wu G, Kanai Y, et al.
FASEB J
. 2013 Dec;
28(3):1294-305.
PMID: 24334703
The mechanisms by which maternal nutrient restriction (MNR) causes reduced fetal growth are poorly understood. We hypothesized that MNR inhibits placental mechanistic target of rapamycin (mTOR) and insulin/IGF-I signaling, down-regulates...