Marc R Reboll
Overview
Explore the profile of Marc R Reboll including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
18
Citations
419
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Wu X, Zheng L, Reboll M, Hyde L, Mass E, Niessen H, et al.
Nat Cardiovasc Res
. 2024 Aug;
3(2):186-202.
PMID: 39196188
Tissue repair after myocardial infarction (MI) is guided by autocrine and paracrine-acting proteins. Deciphering these signals and their upstream triggers is essential when considering infarct healing as a therapeutic target....
2.
Froese N, Szaroszyk M, Galuppo P, Visker J, Werlein C, Korf-Klingebiel M, et al.
J Am Heart Assoc
. 2024 Jan;
13(3):e033553.
PMID: 38293923
Background: Alveolar hypoxia is protective in the context of cardiovascular and ischemic heart disease; however, the underlying mechanisms are incompletely understood. The present study sought to test the hypothesis that...
3.
Reboll M, Klede S, Taft M, Cai C, Field L, Lavine K, et al.
Science
. 2022 Jun;
376(6599):1343-1347.
PMID: 35709278
Effective tissue repair after myocardial infarction entails a vigorous angiogenic response, guided by incompletely defined immune cell-endothelial cell interactions. We identify the monocyte- and macrophage-derived cytokine METRNL (meteorin-like) as a...
4.
Korf-Klingebiel M, Reboll M, Polten F, Weber N, Jackle F, Wu X, et al.
Circulation
. 2021 Aug;
144(15):1227-1240.
PMID: 34372689
Background: Inflammation contributes to the pathogenesis of heart failure, but there is limited understanding of inflammation's potential benefits. Inflammatory cells secrete MYDGF (myeloid-derived growth factor) to promote tissue repair after...
5.
Wu X, Reboll M, Korf-Klingebiel M, Wollert K
Cardiovasc Res
. 2020 Oct;
117(5):1257-1273.
PMID: 33063086
Acute myocardial infarction (MI) inflicts massive injury to the coronary microcirculation leading to vascular disintegration and capillary rarefication in the infarct region. Tissue repair after MI involves a robust angiogenic...
6.
Ebenhoch R, Akhdar A, Reboll M, Korf-Klingebiel M, Gupta P, Armstrong J, et al.
Nat Commun
. 2019 Nov;
10(1):5379.
PMID: 31772377
Myeloid-derived growth factor (MYDGF) is a paracrine-acting protein that is produced by bone marrow-derived monocytes and macrophages to protect and repair the heart after myocardial infarction (MI). This effect can...
7.
Korf-Klingebiel M, Reboll M, Grote K, Schleiner H, Wang Y, Wu X, et al.
Circ Res
. 2019 Aug;
125(9):787-801.
PMID: 31434553
Rationale: Mechanistic insight into the inflammatory response after acute myocardial infarction may inform new molecularly targeted treatment strategies to prevent chronic heart failure. Objective: We identified the sulfatase SULF2 in...
8.
Polten F, Reboll M, Widera C, Kempf T, Bethmann K, Gupta P, et al.
Anal Chem
. 2018 Dec;
91(2):1302-1308.
PMID: 30543396
Myeloid-derived growth factor (MYDGF in humans, Mydgf in mice) is a secreted protein with previously unknown biological functions. In a recent study, Mydgf was shown to mediate cardiac repair after...
9.
Reboll M, Korf-Klingebiel M, Klede S, Polten F, Brinkmann E, Reimann I, et al.
Circulation
. 2017 Sep;
136(19):1809-1823.
PMID: 28931551
Background: Clinical trials of bone marrow cell-based therapies after acute myocardial infarction (MI) have produced mostly neutral results. Treatment with specific bone marrow cell-derived secreted proteins may provide an alternative...
10.
Haddad S, Wang Y, Galy B, Korf-Klingebiel M, Hirsch V, Baru A, et al.
Eur Heart J
. 2016 Aug;
38(5):362-372.
PMID: 27545647
Aims: Iron deficiency (ID) is associated with adverse outcomes in heart failure (HF) but the underlying mechanisms are incompletely understood. Intracellular iron availability is secured by two mRNA-binding iron-regulatory proteins...