M C Morganti-Kossmann
Overview
Explore the profile of M C Morganti-Kossmann including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
40
Citations
1536
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Morganti-Kossmann M, Yan E, Bye N
Injury
. 2010 Apr;
41 Suppl 1:S10-3.
PMID: 20416875
Compared to other neurological diseases, the research surrounding traumatic brain injury (TBI) has a more recent history. The establishment and use of animal models of TBI remains vital to understand...
2.
Grosjean M, Lenzlinger P, Stahel P, Yatsiv I, Shohami E, Trentz O, et al.
Histol Histopathol
. 2006 Dec;
22(3):235-50.
PMID: 17163398
Traumatic brain injury causes progressive tissue atrophy and consequent neurological dysfunction, resulting from neuronal cell death in both animal models and patients. Fas (CD95) and Fas ligand (FasL/CD95L) are important...
3.
Lenzlinger P, Morganti-Kossmann M, Laurer H, McIntosh T
Mol Neurobiol
. 2002 Feb;
24(1-3):169-81.
PMID: 11831551
One and a half to two million people sustain a traumatic brain injury (TBI) in the US each year, of which approx 70,000-90,000 will suffer from long-term disability with dramatic...
4.
Morganti-Kossmann M, Rancan M, Otto V, Stahel P, Kossmann T
Shock
. 2001 Sep;
16(3):165-77.
PMID: 11531017
Neuroinflammation occuring after traumatic brain injury (TBI) is a complex phenomenon comprising distinct cellular and molecular events involving the injured as well as the healthy cerebral tissue. Although immunoactivation only...
5.
Stahel P, Morganti-Kossmann M, Perez D, Redaelli C, Gloor B, Trentz O, et al.
J Neurotrauma
. 2001 Aug;
18(8):773-81.
PMID: 11526983
It has become evident in recent years that intracranial inflammation after traumatic brain injury (TBI) is, at least in part, mediated by activation of the complement system. However, most conclusions...
6.
Otto V, Stahel P, Rancan M, Kariya K, Shohami E, Yatsiv I, et al.
Neuroreport
. 2001 Jul;
12(9):2059-64.
PMID: 11435946
The expression of the chemokines macrophage inflammatory protein (MIP)-2 and MIP-1alpha and of their receptors CXCR2 and CCR5 was assessed in wild type (WT) and TNF/lymphotoxin-alpha knockout (TNF/LT-alpha-/-) mice subjected...
7.
Pleines U, Morganti-Kossmann M, Rancan M, Joller H, Trentz O, Kossmann T
J Neurotrauma
. 2001 Jun;
18(5):491-8.
PMID: 11393252
It has been hypothesized that immunoactivation may contribute to brain damage and affect outcome after traumatic brain injury (TBI). In order to determine the role of inflammation after TBI, we...
8.
Lenzlinger P, Hans V, Joller-Jemelka H, Trentz O, Morganti-Kossmann M, Kossmann T
J Neurotrauma
. 2001 Jun;
18(5):479-89.
PMID: 11393251
The brain is believed to be an immunologically privileged organ, sheltered from the systemic immunological defense by the blood-brain barrier (BBB). However, there is increasing evidence for a marked inflammatory...
9.
Gebhard F, Liener U, Obertacke U, Morganti-Kossmann M, Bruckner U, Kinzl L
Unfallchirurg
. 2001 May;
104(4):365-8.
PMID: 11357704
Recently, in Germany the academic environment has changed and an upheaval occurred that directly do affect academic research activities. Increasingly, the funding of scientific projects is not provided anymore by...
10.
Rancan M, Otto V, Hans V, Gerlach I, Jork R, Trentz O, et al.
J Neurosci Res
. 2001 Feb;
63(5):438-46.
PMID: 11223919
The pathophysiology of traumatic axonal injury (TAI) is only partially understood. In this study, we investigated the inflammatory response as well as the extent of neurological deficit in a rat...