U E Pleines
Overview
Explore the profile of U E Pleines including associated specialties, affiliations and a list of published articles.
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Articles
5
Citations
140
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0
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Recent Articles
1.
Pleines U, Morganti-Kossmann M, Rancan M, Joller H, Trentz O, Kossmann T
J Neurotrauma
. 2001 Jun;
18(5):491-8.
PMID: 11393252
It has been hypothesized that immunoactivation may contribute to brain damage and affect outcome after traumatic brain injury (TBI). In order to determine the role of inflammation after TBI, we...
2.
Stover J, Pleines U, Morganti-Kossmann M, Stocker R, Kossmann T
Crit Care Med
. 1999 Aug;
27(7):1351-7.
PMID: 10446831
Objectives: Brain-injured patients are susceptible to secondary brain damage related to decreased cerebral perfusion pressure associated with edema formation and increased intracranial pressure (ICP). Whenever conventional therapy fails to reduce...
3.
Stover J, Pleines U, Morganti-Kossmann M, Stocker R, Kempski O, Kossmann T
Psychopharmacology (Berl)
. 1999 Feb;
141(1):66-70.
PMID: 9952066
Increased extracellular glutamate levels are related to glial and neuronal damage. Glutamate-mediated toxicity is limited by glial uptake and metabolic transformation of glutamate to glutamine and the energetic compounds alanine...
4.
Pleines U, Stover J, Kossmann T, Trentz O, Morganti-Kossmann M
J Neurotrauma
. 1998 Jun;
15(6):399-409.
PMID: 9624625
The intercellular adhesion molecule-1 (ICAM-1) expressed by endothelial cells is crucial in promoting adhesion and transmigration of circulating leukocytes across the blood-brain barrier (BBB). Migrated immunocompetent cells, in turn, release...
5.
Stover J, Pleines U, Morganti-Kossmann M, Kossmann T, Lowitzsch K, Kempski O
Eur J Clin Invest
. 1998 Feb;
27(12):1038-43.
PMID: 9466133
The excitatory transmitters glutamate and aspartate become toxic whenever their extracellular levels are increased because of neuronal, glial and endothelial impairment. Taurine, a volume-regulating amino acid, is released upon excitotoxin-induced...