M A Pappolla
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Explore the profile of M A Pappolla including associated specialties, affiliations and a list of published articles.
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53
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1919
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Recent Articles
1.
Pappolla M, Matsubara E, Vidal R, Pacheco-Quinto J, Poeggeler B, Zagorski M, et al.
Curr Alzheimer Res
. 2018 Apr;
15(7):637-642.
PMID: 29637859
Background: It has been postulated that inadequate clearance of the amyloid β protein (Aβ) plays an important role in the accumulation of Aβ in sporadic late onset Alzheimer's disease (AD)....
2.
Matsubara E, Tsuchiya A, Minami N, Nishino I, Pappolla M, Shoji M, et al.
Eur J Neurol
. 2007 Jun;
14(7):819-22.
PMID: 17594342
A unique sib pair afflicted by limb girdle muscular dystrophy type 2A (LGMD2A) is described showing a slowly progressive autosomal recessive type of muscular dystrophy with onset in the third...
3.
Mild hypercholesterolemia is an early risk factor for the development of Alzheimer amyloid pathology
Pappolla M, Bryant-Thomas T, Herbert D, Pacheco J, Fabra Garcia M, Manjon M, et al.
Neurology
. 2003 Jul;
61(2):199-205.
PMID: 12874399
Background: Epidemiologic and experimental data suggest that cholesterol may play a role in the pathogenesis of AD. Modulation of cholesterolemia in transgenic animal models of AD strongly alters amyloid pathology....
4.
Poeggeler B, Miravalle L, Zagorski M, Wisniewski T, Chyan Y, Zhang Y, et al.
Biochemistry
. 2001 Dec;
40(49):14995-5001.
PMID: 11732920
Inheritance of apoE4 is a strong risk factor for the development of late-onset sporadic Alzheimer's disease (AD). Several lines of evidence suggest that apoE4 binds to the Alzheimer Abeta protein...
5.
Refolo L, Pappolla M, Lafrancois J, Malester B, Schmidt S, Tint G, et al.
Neurobiol Dis
. 2001 Oct;
8(5):890-9.
PMID: 11592856
Clinical, epidemiological, and laboratory studies suggest that cholesterol may play a role in the pathogenesis of Alzheimer's disease (AD). Transgenic mice exhibiting an Alzheimer's beta-amyloid phenotype were treated with the...
6.
Andorn A, Pappolla M
Free Radic Biol Med
. 2001 Jul;
31(3):315-20.
PMID: 11461768
Some catecholamines and indolamines inhibit lipid peroxidation. Recent studies indicate that catecholaminergic inhibition of lipid peroxidation may be receptor mediated in vivo and in cell cultures. Because oxidative stress is...
7.
Pappolla M, Omar R, Chyan Y, Ghiso J, Hsiao K, Bozner P, et al.
J Neurochem
. 2001 Jul;
78(1):121-8.
PMID: 11432979
A large body of data suggests that the Alzheimer's amyloid peptide (Abeta) causes degeneration and death of neurons by mechanisms that involve reactive oxygen species. The pathways involved in Abeta-mediated...
8.
9.
Nunomura A, Perry G, Pappolla M, Friedland R, Hirai K, Chiba S, et al.
J Neuropathol Exp Neurol
. 2000 Nov;
59(11):1011-7.
PMID: 11089579
The predictable chronological sequence of pathological events in Down syndrome (DS) provides the opportunity to rigorously investigate the relationship between oxidative stress and amyloid-beta (Abeta) deposition. In this study, we...
10.
Refolo L, Malester B, Lafrancois J, Wang R, Tint G, Sambamurti K, et al.
Neurobiol Dis
. 2000 Aug;
7(4):321-31.
PMID: 10964604
Recent data suggest that cholesterol metabolism is linked to susceptibility to Alzheimer's disease (AD). However, no direct evidence has been reported linking cholesterol metabolism and the pathogenesis of AD. To...