Lori N Griner
Overview
Explore the profile of Lori N Griner including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
6
Citations
167
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Sharma G, Guardia C, Roy A, Vassilev A, Saric A, Griner L, et al.
Autophagy
. 2019 Feb;
15(10):1694-1718.
PMID: 30806145
High-throughput screening identified 5 chemical analogs (termed the WX8-family) that disrupted 3 events in lysosome homeostasis: (1) lysosome fission via tubulation without preventing homotypic lysosome fusion; (2) trafficking of molecules...
2.
Basiorka A, McGraw K, De Ceuninck L, Griner L, Zhang L, Clark J, et al.
Cancer Res
. 2016 May;
76(12):3531-40.
PMID: 27197154
In a subset of patients with non-del(5q) myelodysplastic syndrome (MDS), lenalidomide promotes erythroid lineage competence and effective erythropoiesis. To determine the mechanism by which lenalidomide promotes erythropoiesis, we investigated its...
3.
Mazzacurati L, Lambert Q, Pradhan A, Griner L, Huszar D, Reuther G
Oncotarget
. 2015 Oct;
6(37):40141-57.
PMID: 26472029
Classical myeloproliferative neoplasms (MPNs) are hematopoietic stem cell disorders that exhibit excess mature myeloid cells, bone marrow fibrosis, and risk of leukemic transformation. Aberrant JAK2 signaling plays an etiological role...
4.
Griner L, McGraw K, Johnson J, List A, Reuther G
Br J Haematol
. 2012 Nov;
160(2):177-87.
PMID: 23157224
Aberrant JAK2 signalling plays an important role in the aetiology of myeloproliferative neoplasms (MPNs). JAK2 inhibitors, however, do not readily eliminate neoplastic MPN cells and thus do not induce patient...
5.
Griner L, Reuther G
Cancer Biol Ther
. 2010 Nov;
10(10):979-82.
PMID: 21084860
Chronic myeloid leukemia (CML) progresses from a chronic phase to a deadly blast crisis phase. While it is known that BCR-ABL initiates the disease and that secondary molecular and genetic...
6.
Pradhan A, Lambert Q, Griner L, Reuther G
J Biol Chem
. 2010 Apr;
285(22):16651-63.
PMID: 20363735
The JAK2-V617F mutation is an important etiologic factor for the development of myeloproliferative neoplasms. The mechanism by which this mutated tyrosine kinase initiates deregulated signals in cells is not completely...