Kurt Jungermann
Overview
Explore the profile of Kurt Jungermann including associated specialties, affiliations and a list of published articles.
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Articles
12
Citations
184
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Recent Articles
1.
Pestel S, Jungermann K, Schieferdecker H
Prostaglandins Other Lipid Mediat
. 2005 Mar;
75(1-4):123-39.
PMID: 15789620
In contrast to conventionally used immunoassays, thin layer chromatography (TLC)--by prelabeling of cells with radioactive arachidonic acid (AA)--allows to differentiate between cellularly built and added prostanoids and thus to investigate...
2.
Roth U, Jungermann K, Kietzmann T
Biol Chem
. 2004 May;
385(3-4):239-47.
PMID: 15134337
Glucokinase (GK) is the key enzyme of glucose utilization in liver and is localized in the less aerobic perivenous area. Until now, the O2-responsive elements in the liver-specific GK promoter...
3.
Liu Q, Berchner-Pfannschmidt U, Moller U, Brecht M, Wotzlaw C, Acker H, et al.
Proc Natl Acad Sci U S A
. 2004 Mar;
101(12):4302-7.
PMID: 15010533
It has been proposed that hydroxyl radicals (.OH) generated in a perinuclear iron-dependent Fenton reaction are involved in O(2)-dependent gene expression. Thus, it was the aim of this study to...
4.
Pestel S, Schlaf G, Gotze O, Jungermann K, Schieferdecker H
Lab Invest
. 2003 Dec;
83(12):1733-41.
PMID: 14691291
Various inflammatory stimuli such as anaphylatoxin C5a, zymosan, and lipopolysaccharides (LPSs) have been reported both to enhance glucose output in the perfused rat liver and to induce prostanoid (ie, prostaglandin...
5.
Pestel S, Nath A, Jungermann K, Schieferdecker H
Biochem Pharmacol
. 2003 Aug;
66(4):643-52.
PMID: 12906929
Prostanoids, i.e. prostaglandins and thromboxane, regulate liver-specific functions both in homeostasis and during defense reactions. For example, prostanoids are released from Kupffer cells, the resident liver macrophages, in response to...
6.
Gorlach A, Berchner-Pfannschmidt U, Wotzlaw C, Cool R, Fandrey J, Acker H, et al.
Thromb Haemost
. 2003 Apr;
89(5):926-35.
PMID: 12719791
The hypoxia-inducible transcription factor HIF-1 mediates upregulation of plasminogen activator inhibitor-1 (PAI-1) expression under hypoxia. Reactive oxygen species (ROS) have also been implicated in PAI-1 gene expression. However, the role...
7.
Kietzmann T, Jungermann K, Gorlach A
Thromb Haemost
. 2003 Apr;
89(4):666-73.
PMID: 12669121
Mitogen-activated protein kinases (MAPKs) and protein kinase B (PKB) mediate growth and stress signals and have been implicated in the hypoxic response. Under hypoxic conditions, the expression of plasminogen activator...
8.
Kietzmann T, Samoylenko A, Roth U, Jungermann K
Blood
. 2002 Oct;
101(3):907-14.
PMID: 12393531
The expression of the plasminogen activator inhibitor-1 (PAI-1) gene is enhanced by insulin both in vivo and in various cell types. Because insulin exerts a number of its biologic activities...
9.
Kietzmann T, Krones-Herzig A, Jungermann K
Biochem Pharmacol
. 2002 Sep;
64(5-6):903-11.
PMID: 12213585
The substrates oxygen and glucose are important for the appropriate regulation of metabolism, angiogenesis, tumorigenesis and embryonic development. The knowledge about an interaction between these two signals is limited. We...
10.
Pestel S, Jungermann K, Gotze O, Schieferdecker H
Lab Invest
. 2002 Apr;
82(4):463-71.
PMID: 11950903
The proinflammatory anaphylatoxin C5a induces the release of prostanoids, ie, prostaglandins (PG) and thromboxane (TX), from the resident liver macrophages (Kupffer cells [KC]). Because KC themselves express prostanoid receptors, prostanoids--besides...