Keith W Dilly
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Explore the profile of Keith W Dilly including associated specialties, affiliations and a list of published articles.
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9
Citations
718
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Recent Articles
1.
van Olden R, Lo Bianco C, Dilly K, Savelieva M, Xu S, Tijsma A, et al.
Mol Ther Methods Clin Dev
. 2025 Feb;
32(4):101344.
PMID: 40018025
Gene therapies such as onasemnogene abeparvovec for spinal muscular atrophy (SMA) utilize adeno-associated virus 9 (AAV9) for targeted gene delivery, which requires an AAV9 antibody (AAV9-Ab) immunoglobulin G (IgG) ≤1:50...
2.
McGrattan K, Walsh K, Mehl L, Kaur S, Dilly K
J Neuromuscul Dis
. 2025 Feb;
:22143602241303373.
PMID: 39973401
Background: Improvement and maintenance of bulbar function are important goals of disease-modifying treatments (DMTs) for spinal muscular atrophy (SMA), but standardized and validated measures for assessing bulbar function do not...
3.
Day J, Mendell J, Burghes A, van Olden R, Adhikary R, Dilly K
Mol Ther Methods Clin Dev
. 2023 Oct;
31:101117.
PMID: 37822718
Onasemnogene abeparvovec is a recombinant adeno-associated virus serotype 9 (AAV9) vector-based gene therapy for spinal muscular atrophy (SMA). Patients with elevated titers of anti-AAV9 antibodies (AAV9-Ab) should not receive onasemnogene...
4.
Rossow C, Dilly K, Yuan C, Nieves-Cintron M, Cabarrus J, Santana L
J Mol Cell Cardiol
. 2008 Nov;
46(2):249-56.
PMID: 19027024
In heart, pore-forming Kv4 alpha channel subunits underlie the K(+) transient outward current (I(to)). Expression of Kv4 is greater in left ventricular epicardial (EPI) than in endocardial (ENDO) cells, resulting...
5.
Santana L, Nunez-Duran H, Dilly K, Lederer W
Heart Fail Clin
. 2007 Mar;
1(2):193-205.
PMID: 17386846
No abstract available.
6.
Rossow C, Dilly K, Santana L
Circ Res
. 2006 Apr;
98(10):1306-13.
PMID: 16614306
Kv4 channels are differentially expressed across the mouse left ventricular free wall. Accordingly, the transient outward K+ current (Ito), which is produced by Kv4 channels, is greater in left ventricular...
7.
Dilly K, Rossow C, Votaw V, Meabon J, Cabarrus J, Santana L
J Physiol
. 2006 Jan;
572(Pt 1):227-41.
PMID: 16423856
Ca(2+) release during excitation-contraction (EC) coupling varies across the left ventricular free wall. Here, we investigated the mechanisms underlying EC coupling differences between mouse left ventricular epicardial (Epi) and endocardial...
8.
Dilly K, Kurokawa J, Terrenoire C, Reiken S, Lederer W, Marks A, et al.
J Biol Chem
. 2004 Jul;
279(39):40778-87.
PMID: 15272004
The cardiac slow delayed rectifier potassium channel (IKs), comprised of (KCNQ1) and beta (KCNE1) subunits, is regulated by sympathetic nervous stimulation, with activation of beta-adrenergic receptors PKA phosphorylating IKs channels....
9.
Mohler P, Schott J, Gramolini A, Dilly K, Guatimosim S, Dubell W, et al.
Nature
. 2003 Feb;
421(6923):634-9.
PMID: 12571597
Mutations in ion channels involved in the generation and termination of action potentials constitute a family of molecular defects that underlie fatal cardiac arrhythmias in inherited long-QT syndrome. We report...
10.
Choi K, Zhong Y, Hoit B, Grupp I, Hahn H, Dilly K, et al.
Am J Physiol Heart Circ Physiol
. 2002 Sep;
283(4):H1398-408.
PMID: 12234790
The goal of the study was to determine whether defects in intracellular Ca(2+) signaling contribute to cardiomyopathy in streptozotocin (STZ)-induced diabetic rats. Depression in cardiac systolic and diastolic function was...