Kayalvizhi Radhakrishnan
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Explore the profile of Kayalvizhi Radhakrishnan including associated specialties, affiliations and a list of published articles.
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8
Citations
102
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Recent Articles
1.
Radhakrishnan K, Zhang Y, Mustapha O, Weigel T, Upchurch C, Upchurch C, et al.
bioRxiv
. 2025 Jan;
PMID: 39868327
Oxidative stress is a prominent feature of Alzheimer's disease. Within this context, cholesterol undergoes oxidation, producing the pro-inflammatory product 7-ketocholesterol (7-KC). In this study, we observe elevated levels of 7-KC...
2.
Kamp M, Shakeri B, Tevoufouet E, Krieger A, Henry M, Behnke K, et al.
Biochim Biophys Acta
. 2012 May;
1824(9):1045-57.
PMID: 22633975
Ca(v)2.3 containing voltage-activated Ca(2+) channels are expressed in excitable cells and trigger neurotransmitter and peptide-hormone release. Their expression remote from the fast release sites leads to the accumulation of presynaptic...
3.
Radhakrishnan K, Krieger A, Dibue M, Hescheler J, Schneider T
Cell Physiol Biochem
. 2011 Dec;
28(4):603-12.
PMID: 22178872
Background: Voltage gated calcium channels (VGCCs) regulate cellular activity in response to membrane depolarization by altering calcium homeostasis. Because calcium is the most versatile second messenger, regulation of the amount...
4.
Radhakrishnan K, Kamp M, Siapich S, Hescheler J, Luke M, Schneider T
Cell Physiol Biochem
. 2011 Jun;
27(5):421-32.
PMID: 21691059
Background: Calcium channels are essential in coupling action potential to signal transduction in cells. There are several types of calcium channels, which can be pharmacologically classified as L-, N-, P/Q-,...
5.
Radhakrishnan K, Hescheler J, Schneider T
Curr Pharm Biotechnol
. 2010 Feb;
11(2):175-9.
PMID: 20166964
Screening for protein interaction partners of ion channels helps to elucidate signaling cascades to cellular targets and processes for a better understanding of the origin of diseases. Most important are...
6.
Weiergraber M, Henry M, Radhakrishnan K, Hescheler J, Schneider T
J Neurophysiol
. 2007 Mar;
97(5):3660-9.
PMID: 17376845
Voltage-gated calcium channels are key components in the etiology and pathogenesis of epilepsies. Former studies mainly focused on P/Q-type Ca(v)2.1 and T-type Ca(v)3.2 Ca(2+) channels involved in absence epileptogenesis, but...
7.
Weiergraber M, Kamp M, Radhakrishnan K, Hescheler J, Schneider T
Neurosci Biobehav Rev
. 2006 Sep;
30(8):1122-44.
PMID: 16963121
The Ca(v)2.3 encoded Ca2+ channel is probably one of the least well-understood voltage-gated calcium channels in terms of physiology, pharmacology and clinical relevance. Here we provide a detailed insight into...
8.
Weiergraber M, Henry M, Krieger A, Kamp M, Radhakrishnan K, Hescheler J, et al.
Epilepsia
. 2006 May;
47(5):839-50.
PMID: 16686648
Purpose: Recently the Ca(v)2.3 (E/R-type) voltage-gated calcium channel (VGCC) has turned out to be not only a potential target for different antiepileptic drugs (e.g., lamotrigine, topiramate) but also a crucial...
9.
Krieger A, Radhakrishnan K, Pereverzev A, Siapich S, Banat M, Kamp M, et al.
Cell Physiol Biochem
. 2006 Mar;
17(3-4):97-110.
PMID: 16543726
Multiple types of voltage-activated Ca2+ channels (T, L, N, P, Q, R type) coexist in excitable cells and participate in synaptic differentiation, secretion, transmitter release, and neuronal plasticity. Ca2+ ions...