Julio C Resendiz
Overview
Explore the profile of Julio C Resendiz including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
9
Citations
86
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Lepantalo A, Virtanen K, Resendiz J, Mikkelsson J, Viiri L, Karhunen P, et al.
Thromb Res
. 2009 Feb;
124(2):193-8.
PMID: 19232433
Introduction: Large individual variability in clopidogrel responses has been reported. However, mechanisms of the non-responsiveness are unclear. Our aim was to study the extent of platelet inhibition at the receptor...
2.
Forestier M, Resendiz J, Pontiggia L, Lassila R, Beer J
Blood Coagul Fibrinolysis
. 2008 Nov;
19(8):801-6.
PMID: 19002047
Our aim was to investigate the role of filamin cleavage and protein tyrosine phosphorylation in shear-stress-induced platelet microparticle formation and of its suppression by the monoclonal antibody (mAb) Ib-23 directed...
3.
Mayranpaa M, Resendiz J, Heikkila H, Lindstedt K, Kovanen P
Endothelium
. 2007 May;
14(2):81-7.
PMID: 17497364
Loss of endothelial cells (ECs) with ensuing exposure of thrombogenic subendothelial surface is a common cause of thromboembolic complications in atherosclerotic arteries. Thus, endothelial denudation has emerged as a major...
4.
Feng S, Lu X, Resendiz J, Kroll M
Am J Physiol Cell Physiol
. 2006 Jul;
291(6):C1346-54.
PMID: 16822941
Integrin mechanotransduction is a ubiquitous biological process. Mechanical forces are transduced transmembranously by an integrin's ligand-bound extracellular domain through its beta-subunit's cytoplasmic domain connected to the cytoskeleton. This often culminates...
5.
Lepantalo A, Mikkelsson J, Resendiz J, Viiri L, Backman J, Kankuri E, et al.
Thromb Haemost
. 2006 Feb;
95(2):253-9.
PMID: 16493486
The antiplatelet effect of aspirin varies individually. This study evaluated whether the antiplatelet effect of aspirin associates with polymorphisms in the genes coding for cyclo-oxygenase-1 (COX-1) and several platelet glycoprotein...
6.
Sun L, Feng S, Resendiz J, Lu X, Durante W, Kroll M
Ann Biomed Eng
. 2004 Oct;
32(9):1193-201.
PMID: 15493507
Mechanisms of shear-induced platelet aggregation are not established. Data that ristocetin-induced von Willebrand factor (VWF) binding to glycoprotein (Gp) Ibalpha activates proline-rich tyrosine kinase 2 (Pyk2) and extracellular-regulated kinase (ERK)...
7.
Feng S, Resendiz J, Lu X, Kroll M
Blood
. 2003 Jun;
102(6):2122-9.
PMID: 12791664
We examined the hypothesis that filamin A binding to the cytoplasmic tail of platelet glycoprotein Ibalpha (GpIbalpha) is regulated by pathologic shear stress and modulates von Willebrand factor (VWF)-induced platelet...
8.
Resendiz J, Feng S, Ji G, Francis K, Berndt M, Kroll M
Mol Pharmacol
. 2003 Feb;
63(3):639-45.
PMID: 12606772
Pathologically elevated shear stress triggers aspirin-insensitive platelet thrombosis. Signaling mechanisms involved in shear-induced platelet thrombosis are not well understood. To investigate these, we examined the hypothesis that functionally important platelet...
9.
Feng S, Resendiz J, Christodoulides N, Lu X, Arboleda D, Berndt M, et al.
Biochemistry
. 2002 Jan;
41(4):1100-8.
PMID: 11802708
Shear-induced platelet responses are triggered by VWF binding to the platelet GpIb-IX complex, and there is evidence that this ligand-receptor coupling stimulates transmembranous signaling through the cytoplasmic tail of glycoprotein...