Juliane Mohr
Overview
Explore the profile of Juliane Mohr including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
23
Citations
413
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Zou M, Pezoldt J, Mohr J, Philipsen L, Leufgen A, Cerovic V, et al.
Cell Rep
. 2024 Apr;
43(5):114153.
PMID: 38687643
Gut-draining mesenteric and celiac lymph nodes (mLNs and celLNs) critically contribute to peripheral tolerance toward food and microbial antigens by supporting the de novo induction of regulatory T cells (Tregs)....
2.
Baars I, Jaedtka M, Dewitz L, Fu Y, Franz T, Mohr J, et al.
JCI Insight
. 2023 Jun;
8(14).
PMID: 37310793
The virulence of intracellular pathogens relies largely on the ability to survive and replicate within phagocytes but also on release and transfer into new host cells. Such cell-to-cell transfer could...
3.
Formaglio P, Alabdullah M, Siokis A, Handschuh J, Sauerland I, Fu Y, et al.
Immunity
. 2021 Oct;
54(12):2724-2739.e10.
PMID: 34687607
Nitric oxide (NO) is an important antimicrobial effector but also prevents unnecessary tissue damage by shutting down the recruitment of monocyte-derived phagocytes. Intracellular pathogens such as Leishmania major can hijack...
4.
Kleinholz C, Riek-Burchardt M, Seiss E, Amore J, Gintschel P, Philipsen L, et al.
Sci Rep
. 2021 Jul;
11(1):15071.
PMID: 34302006
Neutrophils represent one of the first immune cell types recruited to sites of infection, where they can control pathogens by phagocytosis and cytotoxic mechanisms. Intracellular pathogens such as Leishmania major...
5.
Lucas B, Riebau C, Mohr J, Pliske G, Walcher F, Piatek S
BMC Musculoskelet Disord
. 2021 Jan;
22(1):113.
PMID: 33499843
Background: One of the most common complications of hip arthroplasty is excessive blood loss that could necessitate allogenic blood transfusion, which is further associated with other complications, such as infections,...
6.
Jayavelu A, Schnoder T, Perner F, Herzog C, Meiler A, Krishnamoorthy G, et al.
Nature
. 2020 Nov;
588(7836):157-163.
PMID: 33239784
Janus kinases (JAKs) mediate responses to cytokines, hormones and growth factors in haematopoietic cells. The JAK gene JAK2 is frequently mutated in the ageing haematopoietic system and in haematopoietic cancers....
7.
Knop L, Deiser K, Bank U, Witte A, Mohr J, Philipsen L, et al.
Eur J Immunol
. 2020 Feb;
50(6):846-857.
PMID: 32043573
The survival of peripheral T cells is dependent on their access to peripheral LNs (pLNs) and stimulation by IL-7. In pLNs fibroblastic reticular cells (FRCs) and lymphatic endothelial cells (LECs)...
8.
Dash B, Schnoder T, Kathner C, Mohr J, Weinert S, Herzog C, et al.
J Cancer Res Clin Oncol
. 2018 Aug;
144(10):1933-1944.
PMID: 30083817
Purpose: Cell fate determinants Scrib and Llgl1 influence self-renewal capacity of hematopoietic stem cells (HSCs). Scrib-deficient HSCs are functionally impaired and lack sufficient repopulation capacity during serial transplantation and stress....
9.
Edelmann B, Gupta N, Schnoeder T, Oelschlegel A, Shahzad K, Goldschmidt J, et al.
J Clin Invest
. 2018 Jul;
128(10):4359-4371.
PMID: 30024857
JAK2-V617F-positive chronic myeloproliferative neoplasia (CMN) commonly displays dysfunction of integrins and adhesion molecules expressed on platelets, erythrocytes, and leukocytes. However, the mechanism by which the 2 major leukocyte integrin chains,...
10.
Mohr J, Dash B, Schnoeder T, Wolleschak D, Herzog C, Santamaria N, et al.
Leukemia
. 2018 Feb;
32(5):1211-1221.
PMID: 29467485
Cell fate determinants influence self-renewal potential of hematopoietic stem cells. Scribble and Llgl1 belong to the Scribble polarity complex and reveal tumor-suppressor function in drosophila. In hematopoietic cells, genetic inactivation...