Jucelia Jeremias Fortunato
Overview
Explore the profile of Jucelia Jeremias Fortunato including associated specialties, affiliations and a list of published articles.
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Articles
23
Citations
257
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Recent Articles
1.
Viana R, Joaquim L, Lippert F, Andrade N, Fleith N, Damasio C, et al.
Microvasc Res
. 2024 Jun;
155:104711.
PMID: 38880383
Ischemic stroke occurs due a blockage in the blood flow to the brain, leading to damage to the nervous system. The prevalent morbidities resulting from stroke include post-stroke infection, as...
2.
Cargnin-Carvalho A, da Silva M, Costa A, Engel N, Farias B, Bressan J, et al.
Biochem Cell Biol
. 2023 Mar;
101(4):313-325.
PMID: 36947832
Excessive fructose consumption is associated with the incidence of obesity and systemic inflammation, resulting in increased oxidative damage and failure to the function of brain structures. Thus, we hypothesized that...
3.
Garbossa L, Joaquim L, Danielski L, Goldim M, Machado R, Metzker K, et al.
Int J Neurosci
. 2022 Nov;
134(8):849-857.
PMID: 36448768
Male Wistar rats (250-350g) were submitted to CLP, or sham as control, and divided into the sham + water, sham + MD (300 mg/kg), CLP + water, and CLP + ...
4.
da Rosa N, Medeiros F, de Oliveira J, Laurentino A, Peretti E, Machado R, et al.
Int J Dev Neurosci
. 2021 Nov;
82(1):39-49.
PMID: 34755374
Objective: 6-Shogaol, bioactive compound of Zingiber officinale Roscoe, has anti-inflammatory, antioxidant, and neuroprotective properties. The objective of the present study was to verify the effect of 6-shogaol on behavioral parameters...
5.
da Rosa N, Medeiros F, de Oliveira J, Laurentino A, Peretti E, Machado R, et al.
Dev Neurosci
. 2021 Oct;
44(1):13-22.
PMID: 34695825
6-Shogaol is one of the main active phenolic components of ginger and has neuroprotective effects by protecting brain against the oxidative stress and regulate the levels of neurotrophic factors. The...
6.
Novochadlo M, Goldim M, Bonfante S, Joaquim L, Mathias K, Metzker K, et al.
Microvasc Res
. 2021 Jun;
137:104193.
PMID: 34062190
Sepsis is a complication of an infection which imbalance the normal regulation of several organ systems, including the central nervous system (CNS). Evidence points towards inflammation and oxidative stress as...
7.
Bonfante S, Joaquim L, Fileti M, Giustina A, Goldim M, Danielski L, et al.
Neurotox Res
. 2020 Oct;
39(2):119-132.
PMID: 33025358
Sepsis-associated encephalopathy is a serious consequence of sepsis, triggered by the host response against an infectious agent, that can lead to brain damage and cognitive impairment. Several mechanisms have been...
8.
Danielski L, Giustina A, Bonfante S, Goldim M, Joaquim L, Metzker K, et al.
Mol Neurobiol
. 2020 Sep;
57(12):5247-5262.
PMID: 32870491
Sepsis survivors present acute and long-term cognitive impairment and the pathophysiology of neurological dysfunction in sepsis involves microglial activation. Recently, the involvement of cytosolic receptors capable of forming protein complexes...
9.
de Farias B, Costa A, Engel N, Goldim M, Turatti C, Cargnin-Cavalho A, et al.
Neurochem Res
. 2020 Aug;
45(10):2487-2498.
PMID: 32789797
Obesity is characterized by chronic inflammation of low grade. The cholinergic anti-inflammatory pathway favors the reduction of the inflammatory response. In this work the effect of stimulation of the cholinergic...
10.
Giustina A, Goldim M, Danielski L, Garbossa L, Junior A, Cidreira T, et al.
Mol Neurobiol
. 2020 Aug;
57(11):4451-4466.
PMID: 32743736
Sepsis causes organ dysfunction due to an infection, and it may impact the central nervous system. Neuroinflammation and oxidative stress are related to brain dysfunction after sepsis. Both processes affect...