Joseph Tripodi
Overview
Explore the profile of Joseph Tripodi including associated specialties, affiliations and a list of published articles.
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28
Citations
553
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Recent Articles
1.
Handa S, Schaniel C, Tripodi J, Ahire D, Mia M, Klingborg S, et al.
Leukemia
. 2024 Dec;
39(3):663-674.
PMID: 39715853
Although multiple genetic events are thought to play a role in promoting progression of the myeloproliferative neoplasms (MPN), the individual events that are associated with the development of more aggressive...
2.
Mutetwa T, Liu Y, Silvera R, Evans M, Yurich M, Tripodi J, et al.
J Acquir Immune Defic Syndr
. 2024 Apr;
96(2):190-195.
PMID: 38630441
Background: People living with HIV (PLWH) have substantially increased incidence of anal precancer and cancer. There are very little data regarding genomic disturbances in anal precancers among PLWH. In this...
3.
Tripodi J, Hoffman R, Tremblay D, Ahire D, Mascarenhas J, Kremyanskaya M, et al.
Int J Mol Sci
. 2024 Apr;
25(7).
PMID: 38612873
The Philadelphia chromosome-negative myeloproliferative neoplasms (Ph-MPNs) are a heterogeneous group of clonal hematopoietic malignancies that include polycythemia vera (PV), essential thrombocythemia (ET), and the prefibrotic form of primary myelofibrosis (prePMF)....
4.
Lanceta J, Tripodi J, Karp L, Shaham M, Mahmood N, Najfeld V, et al.
Mol Cytogenet
. 2023 Nov;
16(1):32.
PMID: 38012697
Lipoblastomas (LPBs) are rare benign neoplasms derived from embryonal adipose that occur predominantly in childhood. LPBs typically present with numeric or structural rearrangements of chromosome 8, the majority of which...
5.
Wang X, Rampal R, Hu C, Tripodi J, Farnoud N, Petersen B, et al.
JCI Insight
. 2022 Mar;
7(8).
PMID: 35259128
Chronic myeloproliferative neoplasms (MPN) frequently evolve to a blast phase (BP) that is almost uniformly resistant to induction chemotherapy or hypomethylating agents. We explored the functional properties, genomic architecture, and...
6.
Mascarenhas J, Kosiorek H, Prchal J, Rambaldi A, Berenzon D, Yacoub A, et al.
Blood
. 2022 Jan;
139(19):2931-2941.
PMID: 35007321
The goal of therapy for patients with essential thrombocythemia (ET) and polycythemia vera (PV) is to reduce thrombotic events by normalizing blood counts. Hydroxyurea (HU) and interferon-α (IFN-α) are the...
7.
Schaniel C, Dhanan P, Hu B, Xiong Y, Raghunandan T, Gonzalez D, et al.
Stem Cell Reports
. 2021 Nov;
16(12):3036-3049.
PMID: 34739849
A library of well-characterized human induced pluripotent stem cell (hiPSC) lines from clinically healthy human subjects could serve as a useful resource of normal controls for in vitro human development,...
8.
Yacoub A, Mascarenhas J, Kosiorek H, Prchal J, Berenzon D, Baer M, et al.
Blood
. 2019 Sep;
134(18):1498-1509.
PMID: 31515250
Prior studies have reported high response rates with recombinant interferon-α (rIFN-α) therapy in patients with essential thrombocythemia (ET) and polycythemia vera (PV). To further define the role of rIFN-α, we...
9.
Vadakke-Madathil S, LaRocca G, Raedschelders K, Yoon J, Parker S, Tripodi J, et al.
Proc Natl Acad Sci U S A
. 2019 May;
116(24):11786-11795.
PMID: 31109997
The extremely limited regenerative potential of adult mammalian hearts has prompted the need for novel cell-based therapies that can restore contractile function in heart disease. We have previously shown the...
10.
Levavi H, Tripodi J, Marcellino B, Mascarenhas J, Jones A, Cross N, et al.
Acta Haematol
. 2019 May;
142(2):105-112.
PMID: 31063994
The JAK2V617F point mutation has been implicated in the pathogenesis of the vast majority of myeloproliferative neoplasms (MPNs), but translocations involving JAK2 have increasingly been identified in patients with JAK2V617F-negativeMPNs....