Joel Abramowitz
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Explore the profile of Joel Abramowitz including associated specialties, affiliations and a list of published articles.
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44
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2060
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Recent Articles
1.
Susperreguy S, Yamashita M, Choi C, Liao Y, Burch L, Blankenship T, et al.
Proc Natl Acad Sci U S A
. 2024 Nov;
121(49):e2411389121.
PMID: 39602257
Using genetically engineered mice and cell lines derived from genetically engineered mice we show that depletion of ER delimited Ca stores activates heteromeric Ca entry (SOCE) channels formed obligatorily, but...
2.
Saliba Y, Jebara V, Hajal J, Maroun R, Chacar S, Smayra V, et al.
Antioxid Redox Signal
. 2018 Oct;
30(16):1851-1879.
PMID: 30318928
Aims: Cardiac fibroblasts (CFs) are emerging as major contributors to myocardial fibrosis (MF), a final common pathway of many etiologies of heart disease. Here, we studied the functional relevance of...
3.
Neuner S, Wilmott L, Hope K, Hoffmann B, Chong J, Abramowitz J, et al.
Behav Brain Res
. 2017 Jun;
332:379.
PMID: 28576459
No abstract available.
4.
Phelan K, Shwe U, Cozart M, Wu H, Mock M, Abramowitz J, et al.
Epilepsia
. 2016 Dec;
58(2):247-254.
PMID: 28012173
Objective: Canonical transient receptor potential (TRPC) channels constitute a family of cation channels that exhibit a regional and cell-specific expression pattern throughout the brain. It has been reported previously that...
5.
Ang R, Abramowitz J, Birnbaumer L, Gourine A, Tinker A
Physiol Rep
. 2016 Aug;
4(15).
PMID: 27528004
The heart is controlled by the sympathetic and parasympathetic limbs of the autonomic nervous system with inhibitory signaling mechanisms recruited in both limbs. The aim of this study was to...
6.
Resistance to pathologic cardiac hypertrophy and reduced expression of CaV1.2 in Trpc3-depleted mice
Han J, Lee Y, Yoen S, Abramowitz J, Birnbaumer L, Lee M, et al.
Mol Cell Biochem
. 2016 Aug;
421(1-2):55-65.
PMID: 27522668
Sustained elevation of intracellular Ca(2+) concentration ([Ca(2+)]i) reprograms cardiovascular cell fate, leading to cellular hypertrophy via Ca(2+)-calmodulin/calcineurin (Cn)/NFAT activation. Accumulating evidence suggests that transient receptor potential canonical (Trpc) channels play...
7.
Sexton J, Desmonds T, Quick K, Taylor R, Abramowitz J, Forge A, et al.
Neurosci Lett
. 2015 Nov;
610:36-42.
PMID: 26520460
Transient receptor potential channels have diverse roles in mechanosensation. Evidence is accumulating that members of the canonical subfamily of TRP channels (TRPC) are involved in touch and hearing. Characteristic features...
8.
Neuner S, Wilmott L, Hope K, Hoffmann B, Chong J, Abramowitz J, et al.
Behav Brain Res
. 2014 Dec;
281:69-77.
PMID: 25513972
Memory formation requires de novo protein synthesis, and memory disorders may result from misregulated synthesis of critical proteins that remain largely unidentified. Plasma membrane ion channels and receptors are likely...
9.
Saliba Y, Karam R, Smayra V, Aftimos G, Abramowitz J, Birnbaumer L, et al.
J Am Soc Nephrol
. 2014 Dec;
26(8):1855-76.
PMID: 25479966
Transient receptor potential canonical (TRPC) Ca(2+)-permeant channels, especially TRPC3, are increasingly implicated in cardiorenal diseases. We studied the possible role of fibroblast TRPC3 in the development of renal fibrosis. In...
10.
Yeon S, Kim J, Yeon D, Abramowitz J, Birnbaumer L, Muallem S, et al.
PLoS One
. 2014 Oct;
9(10):e110413.
PMID: 25310225
Transient receptor potential canonical type 3 (TRPC3) channels are non-selective cation channels and regulate intracellular Ca2+ concentration. We examined the role of TRPC3 channels in agonist-, membrane depolarization (high K+)-,...