Joachim Kirsch
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Explore the profile of Joachim Kirsch including associated specialties, affiliations and a list of published articles.
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55
Citations
1036
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Recent Articles
11.
Kiss E, Groeneweg F, Gorgas K, Schlicksupp A, Kins S, Kirsch J, et al.
J Alzheimers Dis
. 2020 Mar;
74(4):1167-1187.
PMID: 32144981
Early changes in inhibitory synapse connectivities are thought to contribute to the excitation/inhibition imbalance preceding neurodegeneration in Alzheimer's disease (AD). Recently, we reported a robust increase in the level of...
12.
Gaiser M, Kirsch J, Mutzbauer T
J Med Internet Res
. 2020 Feb;
22(4):e15304.
PMID: 32038029
Background: Fatalities rarely occur in dental offices. Implications for clinicians may be deduced from scientific publications and internet reports about deaths in dental offices. Objective: Data involving deaths in dental...
13.
Mustafa R, Kreiner G, Kaminska K, Wood A, Kirsch J, Tucker K, et al.
Front Cell Neurosci
. 2020 Jan;
13:565.
PMID: 31920562
Multiple pathomechanisms triggered by mutant Huntingtin (mHTT) underlie progressive degeneration of dopaminoceptive striatal neurons in Huntington's disease (HD). The primary cilium is a membrane compartment that functions as a hub...
14.
Vetter S, Palesche N, Beisemann N, Schnetzke M, Keil H, Kirsch J, et al.
Eur J Trauma Emerg Surg
. 2020 Jan;
47(4):905-912.
PMID: 31897509
Purpose: The aim of this study was to identify to what extent a dissection of the syndesmosis and an avulsed posterior edge of the tibia can change the tibiofibular diastasis...
15.
Vetter S, Privalov M, Beisemann N, Swartman B, Keil H, Kirsch J, et al.
PLoS One
. 2019 Jun;
14(5):e0217737.
PMID: 31150469
Background: The precise anatomical reduction of the ankle mortise is crucial for the clinical outcome in unstable syndesmotic injuries. Intraoperative cone beam computed tomography (CT), in addition to two-dimensional fluoroscopy,...
16.
Hollnagel J, Elzoheiry S, Gorgas K, Kins S, Beretta C, Kirsch J, et al.
PLoS One
. 2019 Jan;
14(1):e0209228.
PMID: 30645585
Several lines of evidence imply changes in inhibitory interneuron connectivity and subsequent alterations in oscillatory network activities in the pathogenesis of Alzheimer's Disease (AD). Recently, we provided evidence for an...
17.
Groeneweg F, Trattnig C, Kuhse J, Nawrotzki R, Kirsch J
Histochem Cell Biol
. 2018 Sep;
150(5):489-508.
PMID: 30264265
Scaffolding proteins underlying postsynaptic membrane specializations are important structural and functional components of both excitatory and inhibitory synapses. At inhibitory synapses, gephyrin was identified as anchoring protein. Gephyrin self-assembles into...
18.
Obenhaus H, Rozov A, Bertocchi I, Tang W, Kirsch J, Betz H, et al.
Front Mol Neurosci
. 2016 Sep;
9:75.
PMID: 27625595
The causal interrogation of neuronal networks involved in specific behaviors requires the spatially and temporally controlled modulation of neuronal activity. For long-term manipulation of neuronal activity, chemogenetic tools provide a...
19.
Kiss E, Gorgas K, Schlicksupp A, Gross D, Kins S, Kirsch J, et al.
Am J Pathol
. 2016 Jul;
186(9):2279-91.
PMID: 27423698
The pathogenesis of Alzheimer disease (AD) is thought to begin many years before the diagnosis of dementia. Accumulating evidence indicates the involvement of GABAergic neurotransmission in the physiopathology of AD....
20.
OSullivan G, Jedlicka P, Chen H, Kalbouneh H, Ippolito A, Deller T, et al.
Mol Cell Neurosci
. 2016 Feb;
72:101-13.
PMID: 26829712
Mutations that result in the defective trafficking of γ2 subunit containing GABAA receptors (γ2-GABAARs) are known to reduce synaptic inhibition. Whether perturbed clustering of non-mutated GABAARs similarly reduces synaptic inhibition...