Jason Andrew Weesner
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Explore the profile of Jason Andrew Weesner including associated specialties, affiliations and a list of published articles.
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5
Citations
73
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Recent Articles
1.
Fremuth L, Hu H, van de Vlekkert D, Annunziata I, Weesner J, dAzzo A
Cell Rep
. 2025 Jan;
44(1):115204.
PMID: 39817909
Neuraminidase 1 (NEU1) cleaves terminal sialic acids from sialoglycoproteins in endolysosomes and at the plasma membrane. As such, NEU1 regulates immune cells, primarily those of the monocytic lineage. Here, we...
2.
Fremuth L, Hu H, van de Vlekkert D, Annunziata I, Weesner J, Gomero E, et al.
bioRxiv
. 2024 Jun;
PMID: 38826426
Neuraminidase 1 (Neu1) cleaves terminal sialic acids from sialoglycoproteins in endolysosomes and at the plasma membrane. As such, Neu1 regulates immune cells, primarily those of the monocytic lineage. Here we...
3.
Weesner J, Annunziata I, van de Vlekkert D, dAzzo A
FEBS Open Bio
. 2023 Apr;
13(9):1587-1600.
PMID: 37014126
Intracellular organelles carry out many of their functions by engaging in extensive interorganellar communication through specialized membrane contact sites (MCSs) formed where two organelles tether to each other or to...
4.
Weesner J, Annunziata I, Yang T, Acosta W, Gomero E, Hu H, et al.
Cells
. 2022 Aug;
11(16).
PMID: 36010656
GM1-gangliosidosis is a catastrophic, neurodegenerative lysosomal storage disease caused by a deficiency of lysosomal β-galactosidase (β-Gal). The primary substrate of the enzyme is GM1-ganglioside (GM1), a sialylated glycosphingolipid abundant in...
5.
Annunziata I, Weesner J, dAzzo A
Methods Mol Biol
. 2021 Jun;
2277:357-370.
PMID: 34080162
Subcellular fractionation is a valuable procedure in cell biology to separate and purify various subcellular constituents from one another, i.e., nucleus, cytosol, membranes/organelles, and cytoskeleton. The procedure relies on the...
6.
Annunziata I, van de Vlekkert D, Wolf E, Finkelstein D, Neale G, Machado E, et al.
Nat Commun
. 2019 Aug;
10(1):3623.
PMID: 31399583
Coordinated regulation of the lysosomal and autophagic systems ensures basal catabolism and normal cell physiology, and failure of either system causes disease. Here we describe an epigenetic rheostat orchestrated by...