Jane E Tully
Overview
Explore the profile of Jane E Tully including associated specialties, affiliations and a list of published articles.
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10
Citations
310
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Recent Articles
1.
Chapman D, Tully J, Nolin J, Janssen-Heininger Y, Irvin C
J Cell Biochem
. 2014 Jul;
115(12):2055-64.
PMID: 25043224
In a complex inflammatory airways disease such as asthma, abnormalities in a plethora of molecular and cellular pathways ultimately culminate in characteristic impairments in respiratory function. The ability to study...
2.
Nolin J, Tully J, Hoffman S, Guala A, van der Velden J, Poynter M, et al.
Free Radic Biol Med
. 2014 May;
73:143-53.
PMID: 24816292
Interleukin-17A (IL-17A) is a newly emerging player in the pathogenesis of chronic lung diseases that amplifies inflammatory responses and promotes tissue remodeling. Stimulation of lung epithelial cells with IL-17A leads...
3.
van der Velden J, Hoffman S, Alcorn J, Tully J, Chapman D, Lahue K, et al.
Am J Physiol Lung Cell Mol Physiol
. 2014 Mar;
306(9):L866-75.
PMID: 24610935
Chronic allergic asthma leads to airway remodeling and subepithelial fibrosis via mechanisms not fully understood. Airway remodeling is amplified by profibrotic mediators, such as transforming growth factor-β1 (TGF-β1), which plays...
4.
Hoffman S, Tully J, Nolin J, Lahue K, Goldman D, Daphtary N, et al.
Respir Res
. 2013 Dec;
14:141.
PMID: 24364984
Background: The endoplasmic reticulum (ER) stress response participates in many chronic inflammatory and autoimmune diseases. In the current study, we sought to examine the contribution of ER stress transducers in...
5.
Tully J, Hoffman S, Lahue K, Nolin J, Anathy V, Lundblad L, et al.
J Immunol
. 2013 Nov;
191(12):5811-21.
PMID: 24227776
NF-κB activation within the epithelium has been implicated in the pathogenesis of asthma, yet the exact role of epithelial NF-κB in allergen-induced inflammation and airway remodeling remains unclear. In the...
6.
Janssen-Heininger Y, Nolin J, Hoffman S, van der Velden J, Tully J, Lahue K, et al.
J Cell Biochem
. 2013 Apr;
114(9):1962-8.
PMID: 23554102
Glutathione has traditionally been considered as an antioxidant that protects cells against oxidative stress. Hence, the loss of reduced glutathione and formation of glutathione disulfide is considered a classical parameter...
7.
Hoffman S, Tully J, Lahue K, Anathy V, Nolin J, Guala A, et al.
Am J Physiol Lung Cell Mol Physiol
. 2012 Jul;
303(6):L528-38.
PMID: 22752969
Protein-S-glutathionylation (PSSG) is an oxidative modification of reactive cysteines that has emerged as an important player in pathophysiological processes. Under physiological conditions, the thiol transferase, glutaredoxin-1 (Glrx1) catalyses deglutathionylation. Although...
8.
Tully J, Nolin J, Guala A, Hoffman S, Roberson E, Lahue K, et al.
Am J Respir Cell Mol Biol
. 2012 Jun;
47(4):497-508.
PMID: 22652196
The transcription factor NF-κB has been causally linked to inflammatory lung diseases. Recent studies have unraveled the complexity of NF-κB activation by identifying two parallel activation pathways: the classical NF-κB...
9.
Roberson E, Tully J, Guala A, Reiss J, Godburn K, Pociask D, et al.
Am J Respir Cell Mol Biol
. 2011 Jul;
46(5):573-81.
PMID: 21799120
Influenza A virus (IAV) infection is known to induce endoplasmic reticulum (ER) stress, Fas-dependent apoptosis, and TGF-β production in a variety of cells. However, the relationship between these events in...
10.
Aesif S, Kuipers I, van der Velden J, Tully J, Guala A, Anathy V, et al.
Free Radic Biol Med
. 2011 Jul;
51(6):1249-57.
PMID: 21762778
The transcription factor nuclear factor κB (NF-κB) is a critical regulator of inflammation and immunity and is negatively regulated via S-glutathionylation. The inhibitory effect of S-glutathionylation is overcome by glutaredoxin-1...