James D Shull
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Explore the profile of James D Shull including associated specialties, affiliations and a list of published articles.
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31
Citations
651
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Recent Articles
1.
Trentham-Dietz A, Bird J, Gangnon R, Lindberg S, Madison T, Malecki K, et al.
Curr Epidemiol Rep
. 2022 Feb;
9(1):1-9.
PMID: 35223371
Purposeof Review: This review highlights six "best practices" for cancer epidemiology coordinating centers to facilitate the success of a research consortium. Recent Findings: Evidence from emerging literature regarding the Science...
2.
Escanilla N, Hellerstein L, Kleiman R, Kuang Z, Shull J, Page D
Proc Int Conf Mach Learn Appl
. 2019 Dec;
2018:40-47.
PMID: 31799516
There is great interest in methods to improve human insight into trained non-linear models. Leading approaches include producing a ranking of the most relevant features, a non-trivial task for non-linear...
3.
Ding L, Shunkwiler L, Harper N, Zhao Y, Hinohara K, Huh S, et al.
PLoS Genet
. 2019 Mar;
15(3):e1008002.
PMID: 30893315
Mammary epithelial progenitors are the normal cell-of-origin of breast cancer. We previously defined a population of p27+ quiescent hormone-responsive progenitor cells in the normal human breast whose frequency associates with...
4.
Dennison K, Chack A, Hickman M, Harenda Q, Shull J
PLoS One
. 2018 Sep;
13(9):e0204727.
PMID: 30261014
Pituitary adenoma is a common intracranial neoplasm that is observed in approximately 10% of unselected individuals at autopsy. Prolactin-producing adenomas, i.e., prolactinomas, comprise approximately 50% of all pituitary adenomas and...
5.
Jerry D, Shull J, Hadsell D, Rijnkels M, Dunphy K, Schneider S, et al.
Mamm Genome
. 2018 Mar;
29(1-2):24-37.
PMID: 29487996
Breast cancer risk is intimately intertwined with exposure to estrogens. While more than 160 breast cancer risk loci have been identified in humans, genetic interactions with estrogen exposure remain to...
6.
Shull J, Dennison K, Chack A, Trentham-Dietz A
Physiol Genomics
. 2018 Jan;
50(3):215-234.
PMID: 29373076
Numerous laboratory and epidemiologic studies strongly implicate endogenous and exogenous estrogens in the etiology of breast cancer. Data summarized herein suggest that the ACI rat model of 17β-estradiol (E2)-induced mammary...
7.
Das Gupta S, Sae-Tan S, Wahler J, So J, Bak M, Cheng L, et al.
Cancer Prev Res (Phila)
. 2015 Jul;
8(9):807-16.
PMID: 26130252
This study evaluated the anticancer activity and mechanism of action of a γ-tocopherol-rich tocopherol mixture, γ-TmT, in two different animal models of estrogen-induced breast cancer. The chemopreventive effect of γ-TmT...
8.
Dennison K, Samanas N, Harenda Q, Hickman M, Seiler N, Ding L, et al.
Endocr Relat Cancer
. 2015 Mar;
22(2):239-48.
PMID: 25800038
The ACI rat model of 17β-estradiol (E2)-induced mammary cancer is highly relevant for use in establishing the endocrine, genetic, and environmental bases of breast cancer etiology and identifying novel agents...
9.
Samanas N, Commers T, Dennison K, Harenda Q, Kurz S, Lachel C, et al.
PLoS One
. 2015 Feb;
10(2):e0118147.
PMID: 25693193
Congenital anomalies of the kidney and urogenital tract (CAKUT) occur in approximately 0.5% of live births and represent the most frequent cause of end-stage renal disease in neonates and children....
10.
Flister M, Endres B, Rudemiller N, Sarkis A, Santarriaga S, Roy I, et al.
Cancer Res
. 2014 Aug;
74(22):6419-29.
PMID: 25172839
The majority of causative variants in familial breast cancer remain unknown. Of the known risk variants, most are tumor cell autonomous, and little attention has been paid yet to germline...