J W Halliday
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Explore the profile of J W Halliday including associated specialties, affiliations and a list of published articles.
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159
Citations
1508
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Recent Articles
1.
Dodd P, Eckert A, Fletcher L, Kril J, Harper C, Halliday J
Addict Biol
. 2016 Jan;
2(3):337-48.
PMID: 26735790
Transferrin (T f) and its carbohydrate-deficient isoform (CDT) were measured by radioimmunoassay in phosphate-buffered saline extracts of two informative areas of cerebral cortex tissue obtained at autopsy from alcoholics without...
2.
Fletcher L, Halliday J
J Intern Med
. 2002 Mar;
251(3):181-92.
PMID: 11886477
Haemochromatosis, a common recessive genetic disorder in people of Northern European descent, is an iron storage disorder characterized by excessive hepatic iron accumulation resulting from disruption of the regulation of...
3.
Fletcher L, Halliday J, Powell L
J Gastroenterol Hepatol
. 1999 Apr;
14(3):202-14.
PMID: 10197487
It is known that the regular consumption of alcohol is responsible for the disruption of normal iron metabolism in humans, resulting in the excess deposition of iron in the liver...
4.
Halliday J
Nutr Rev
. 1998 May;
56(2 Pt 2):s30-7; discussion s54-75.
PMID: 9564175
Although iron is an essential dietary requirement, the amount absorbed by the body is well regulated and depends on body iron stores and on dietary iron availability. There is very...
5.
Anderson G, MURPHY T, Cowley L, Evans B, Halliday J, McLaren G
Genomics
. 1998 Mar;
48(1):34-9.
PMID: 9503013
The sex-linked anemic (sla) mouse carries an anemia that results from an inherited defect of intestinal iron absorption and provides an ideal model with which to investigate this poorly understood...
6.
McLaren C, McLachlan G, Halliday J, Webb S, Leggett B, Jazwinska E, et al.
Gastroenterology
. 1998 Mar;
114(3):543-9.
PMID: 9496946
Background & Aims: An elevated transferrin saturation is the earliest phenotypic abnormality in hereditary hemochromatosis. Determination of transferrin saturation remains the most useful noninvasive screening test for affected individuals, but...
7.
Iancu T, Deugnier Y, Halliday J, Powell L, Brissot P
J Hepatol
. 1997 Nov;
27(4):628-38.
PMID: 9365038
Background/aims: The pathway through which iron contributes to liver cell damage and cirrhosis in genetic hemochromatosis is not clear. The objective of the present study was to describe the ultrastructural...
8.
Bassett M, Leggett B, Halliday J, Webb S, Powell L
J Hepatol
. 1997 Oct;
27(3):517-24.
PMID: 9314130
Aims: To estimate the cost of population screening for haemochromatosis in Australia and to compare the cost of alternative screening strategies. Methods: The costs of screening for haemochromatosis were analysed...
9.
Ooi L, Crawford D, Gotley D, Clouston A, Strong R, Gobe G, et al.
J Hepatol
. 1997 Apr;
26(4):798-807.
PMID: 9126792
Background/aims: The prognosis for patients with hepatocellular carcinoma is poor although tumour encapsulation has been associated with improved survival and disease-free rates. While the source of the tumour capsule is...
10.
Ramm G, Crawford D, Powell L, Walker N, Fletcher L, Halliday J
J Hepatol
. 1997 Mar;
26(3):584-92.
PMID: 9075666
Background/aims: Activated hepatic stellate cells produce increased levels of collagen in animal models of chronic iron overload; however, their role in human genetic haemochromatosis is unknown. This study examined the...