J Schulte am Esch 2nd
Overview
Explore the profile of J Schulte am Esch 2nd including associated specialties, affiliations and a list of published articles.
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10
Citations
163
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Recent Articles
1.
Knoefel W, Alexander A, Tustas R, Schmelzle M, Klein H, Krieg A, et al.
Zentralbl Chir
. 2011 Nov;
138(2):166-72.
PMID: 22086774
Background: The liver has an excellent regenerative capacity after resection. However, below a critical level of future liver remnant volume (FLRV), partial hepatectomy is accompanied by a significant increase of...
2.
Schmelzle M, Peterschulte G, Matthaei H, Schulte am Esch 2nd J, Peiper M, Rehders A, et al.
Zentralbl Chir
. 2011 Jan;
136(1):82-3.
PMID: 21264810
No abstract available.
3.
Schulte am Esch 2nd J, Rogiers X, Robson S
Ann Transplant
. 2002 Mar;
6(3):12-6.
PMID: 11899892
The rejection of xenografts is associated with vascular-based inflammation, thrombocytopenia and the consumption of coagulation factors that may evolve into disseminated intravascular coagulation. Natural regulators of coagulation in porcine xenografts...
4.
Koziak K, Kaczmarek E, Kittel A, Sevigny J, Blusztajn J, Schulte am Esch 2nd J, et al.
J Biol Chem
. 2000 Jan;
275(3):2057-62.
PMID: 10636909
Ectonucleotidases influence purinergic receptor function by the hydrolysis of extracellular nucleotides. CD39 is an integral membrane protein that is a prototype member of the nucleoside 5'-triphosphate diphosphohydrolase family. The native...
5.
Imai M, Kaczmarek E, Koziak K, Sevigny J, Goepfert C, Guckelberger O, et al.
Biochemistry
. 1999 Oct;
38(41):13473-9.
PMID: 10521254
Vascular ATP diphosphohydrolase/CD39 is an endothelial cell membrane protein with both ecto-ATPase and ecto-ADPase activities. Suppression of constitutive CD39 expression may result in elevated concentrations of ATP and ADP at...
6.
Robson S, Schulte am Esch 2nd J, Bach F
Ann N Y Acad Sci
. 1999 Jul;
875:261-76.
PMID: 10415573
Important mechanisms underlying immediate xenograft loss by hyperacute rejection (HAR), in the pig-to-primate combination, have been recently delineated. There are now several proposed therapies that deal with the problem of...
7.
Schulte am Esch 2nd J, Sevigny J, Kaczmarek E, Siegel J, Imai M, Koziak K, et al.
Biochemistry
. 1999 Feb;
38(8):2248-58.
PMID: 10029517
CD39, the mammalian ATP diphosphohydrolase (ATPDase), is thought to contain two transmembrane domains and five "apyrase conserved regions" (ACR) within a large extracellular region. To study the structure of this...
8.
Kopp C, Grey S, Siegel J, McShea A, Vetr H, Wrighton C, et al.
Transplantation
. 1998 Aug;
66(2):244-51.
PMID: 9701273
Background: Xenograft rejection may predispose to vascular thrombosis because of putative cross-species' functional incompatibilities between natural anticoagulants present on the donor endothelium and host activated coagulation factors. For example, porcine...
9.
Schulte am Esch 2nd J, Cruz M, Siegel J, Anrather J, Robson S
Blood
. 1997 Dec;
90(11):4425-37.
PMID: 9373253
Platelet activation and microthrombus formation are invariable features of xenograft rejection and the vascular injury observed when porcine organs are transplanted into primates. This pathological process could be mediated, at...
10.
Siegel J, Grey S, Lesnikoski B, Kopp C, Soares M, Schulte am Esch 2nd J, et al.
Transplantation
. 1997 Oct;
64(6):888-96.
PMID: 9326416
Background: Delayed xenograft rejection is characterized by platelet activation and fibrin deposition and is thought to occur independently of complement activation. We have therefore investigated the potential for xenogeneic endothelial...