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J M CAILLAUD

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Articles 86
Citations 760
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Recent Articles
1.
Ostos M, Conconi M, Vergnes L, Baroukh N, Ribalta J, Girona J, et al.
Arterioscler Thromb Vasc Biol . 2001 Jun; 21(6):1023-8. PMID: 11397714
Mice expressing human apolipoprotein A-IV (apoA-IV) mainly in the intestine were obtained in an apolipoprotein E-deficient (apoE(0)) background (apoA-IV/E(0) mice). Quantification of aortic lesions and plasma lipid determination showed that...
2.
Boullier A, Hennuyer N, Tailleux A, Furman C, Duverger N, CAILLAUD J, et al.
Clin Sci (Lond) . 2001 Feb; 100(3):343-55. PMID: 11222122
High levels of high-density lipoprotein (HDL) cholesterol have been reported to protect against the development of atherosclerosis in humans by increasing reverse cholesterol transport and inhibiting the oxidation of low-density...
3.
Tailleux A, Bouly M, Luc G, Castro G, CAILLAUD J, Hennuyer N, et al.
Arterioscler Thromb Vasc Biol . 2000 Nov; 20(11):2453-8. PMID: 11073852
Studies performed in vivo have been controversial regarding the implication of human apolipoprotein (apo)A-II in the atherogenic process. Expression of human apoA-II in transgenic mice fed a chow diet leads...
4.
Bettan M, Emmanuel F, Darteil R, CAILLAUD J, Soubrier F, Delaere P, et al.
Mol Ther . 2000 Sep; 2(3):204-10. PMID: 10985950
Numerous diseases are linked to the absence or insufficient concentration of a specific plasma protein. Gene transfer is an appealing strategy for correction of such diseases. We report high and...
5.
Mackness M, Boullier A, Hennuyer N, Mackness B, Hall M, Tailleux A, et al.
Biochem Biophys Res Commun . 2000 Mar; 269(1):232-6. PMID: 10694505
Serum paraoxonase (PON1) is believed to protect against the development of atherosclerosis because of its ability to retard the oxidation of low-density lipoprotein (LDL) by hydrolysing LDL-associated phospholipid and cholesteryl-ester...
6.
Desurmont C, CAILLAUD J, Emmanuel F, Benoit P, Fruchart J, Castro G, et al.
Arterioscler Thromb Vasc Biol . 2000 Feb; 20(2):435-42. PMID: 10669641
The apolipoprotein E (apoE)-deficient mouse is a relevant animal model of human atherosclerosis. Although the prevention of atherosclerosis development has been documented after somatic gene transfer into animal models, regression...
7.
Rouis M, Adamy C, Duverger N, Lesnik P, Horellou P, Moreau M, et al.
Circulation . 1999 Aug; 100(5):533-40. PMID: 10430768
Background: To define the role of metalloproteinases (MMPs) in the development of lipid-rich atherosclerotic lesions in relation to the balance between proteolytic and antiproteolytic activities, we investigated the impact of...
8.
Mir L, Bureau M, Gehl J, Rangara R, Rouy D, CAILLAUD J, et al.
Proc Natl Acad Sci U S A . 1999 Apr; 96(8):4262-7. PMID: 10200250
Gene delivery to skeletal muscle is a promising strategy for the treatment of muscle disorders and for the systemic secretion of therapeutic proteins. However, present DNA delivery technologies have to...
9.
Benoit P, Emmanuel F, CAILLAUD J, Bassinet L, Castro G, Gallix P, et al.
Circulation . 1999 Jan; 99(1):105-10. PMID: 9884386
Background: Apolipoprotein (apo) A-I is the major component of HDL, and it displays antiatherogenic properties. Methods And Results: The human apoA-I gene has been transferred into different mouse models by...
10.
Steg P, Tahlil O, Aubailly N, CAILLAUD J, Dedieu J, Berthelot K, et al.
Circulation . 1997 Jul; 96(2):408-11. PMID: 9244204
Background: Gene delivery of the thymidine kinase (tk) gene combined with ganciclovir (GCV) limits intimal hyperplasia after abrasion of normal arteries. However, the low efficiency of adenoviral-mediated gene transfer to...