J Bart A Crusius
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Explore the profile of J Bart A Crusius including associated specialties, affiliations and a list of published articles.
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2611
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Recent Articles
1.
Cui J, Diogo D, Stahl E, Canhao H, Mariette X, Greenberg J, et al.
Arthritis Rheumatol
. 2016 Oct;
69(4):735-741.
PMID: 27788309
Objective: In many rheumatoid arthritis (RA) patients, disease is controlled with anti-tumor necrosis factor (anti-TNF) biologic therapies. However, in a significant number of patients, the disease fails to respond to...
2.
Wang H, Zhou G, Luo L, Crusius J, Yuan A, Kou J, et al.
Medicine (Baltimore)
. 2015 Oct;
94(42):e1779.
PMID: 26496305
Celiac disease (CD) is common in Caucasians, but thought to be rare in Asians. Our aim was to determine the prevalence of CD in Chinese patients with chronic diarrhea predominant...
3.
Cortes A, Pulit S, Leo P, Pointon J, Robinson P, Weisman M, et al.
Nat Commun
. 2015 May;
6:7146.
PMID: 25994336
Ankylosing spondylitis (AS) is a common, highly heritable, inflammatory arthritis for which HLA-B*27 is the major genetic risk factor, although its role in the aetiology of AS remains elusive. To...
4.
Diogo D, Bastarache L, Liao K, Graham R, Fulton R, Greenberg J, et al.
PLoS One
. 2015 Apr;
10(4):e0122271.
PMID: 25849893
Despite the success of genome-wide association studies (GWAS) in detecting a large number of loci for complex phenotypes such as rheumatoid arthritis (RA) susceptibility, the lack of information on the...
5.
Cortes A, Hadler J, Pointon J, Robinson P, Karaderi T, Leo P, et al.
Nat Genet
. 2013 Jun;
45(7):730-8.
PMID: 23749187
Ankylosing spondylitis is a common, highly heritable inflammatory arthritis affecting primarily the spine and pelvis. In addition to HLA-B*27 alleles, 12 loci have previously been identified that are associated with...
6.
Cui J, Stahl E, Saevarsdottir S, Miceli C, Diogo D, Trynka G, et al.
PLoS Genet
. 2013 Apr;
9(3):e1003394.
PMID: 23555300
Anti-tumor necrosis factor alpha (anti-TNF) biologic therapy is a widely used treatment for rheumatoid arthritis (RA). It is unknown why some RA patients fail to respond adequately to anti-TNF therapy,...
7.
Nijmeijer R, Gadaleta R, van Mil S, van Bodegraven A, Crusius J, Dijkstra G, et al.
PLoS One
. 2011 Sep;
6(8):e23745.
PMID: 21887309
Background: We previously showed that activation of the bile salt nuclear receptor Farnesoid X Receptor (FXR) protects against intestinal inflammation in mice. Reciprocally, these inflammatory mediators may decrease FXR activation....
8.
Weersma R, Crusius J, Roberts R, Koeleman B, Palomino-Morales R, Wolfkamp S, et al.
Inflamm Bowel Dis
. 2010 Sep;
16(12):2080-9.
PMID: 20848524
Background: The Fc receptors II and III (FcgR2a, and FcgR3a) play a crucial role in the regulation of the immune response. The FcgR2a*519GG and FcgR3a*559CC genotypes have been associated with...
9.
Fransen K, Visschedijk M, van Sommeren S, Fu J, Franke L, Festen E, et al.
Hum Mol Genet
. 2010 Jul;
19(17):3482-8.
PMID: 20601676
Genome-wide association studies (GWAS) for Crohn's disease (CD) have identified loci explaining approximately 20% of the total genetic risk of CD. Part of the other genetic risk loci is probably...
10.
Stahl E, Raychaudhuri S, Remmers E, Xie G, Eyre S, Thomson B, et al.
Nat Genet
. 2010 May;
42(6):508-14.
PMID: 20453842
To identify new genetic risk factors for rheumatoid arthritis, we conducted a genome-wide association study meta-analysis of 5,539 autoantibody-positive individuals with rheumatoid arthritis (cases) and 20,169 controls of European descent,...