Ivy Cuijt
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Explore the profile of Ivy Cuijt including associated specialties, affiliations and a list of published articles.
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15
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1880
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Recent Articles
1.
Wauters E, Van Mossevelde S, Sleegers K, van der Zee J, Engelborghs S, Sieben A, et al.
Neurobiol Aging
. 2018 Apr;
67:84-94.
PMID: 29653316
We previously reported a granulin (GRN) null mutation, originating from a common founder, in multiple Belgian families with frontotemporal dementia. Here, we used data of a 10-year follow-up study to...
2.
Boeynaems S, Bogaert E, Michiels E, Gijselinck I, Sieben A, Jovicic A, et al.
Sci Rep
. 2016 Feb;
6:20877.
PMID: 26869068
Hexanucleotide repeat expansions in C9orf72 are the most common cause of amyotrophic lateral sclerosis (ALS) and frontotemporal degeneration (FTD) (c9ALS/FTD). Unconventional translation of these repeats produces dipeptide repeat proteins (DPRs)...
3.
Gijselinck I, Van Mossevelde S, van der Zee J, Sieben A, Philtjens S, Heeman B, et al.
Neurology
. 2015 Nov;
85(24):2116-25.
PMID: 26581300
Objective: To assess the genetic contribution of TBK1, a gene implicated in amyotrophic lateral sclerosis (ALS), frontotemporal dementia (FTD), and FTD-ALS, in Belgian FTD and ALS patient cohorts containing a...
4.
Janssens J, Philtjens S, Kleinberger G, Van Mossevelde S, van der Zee J, Cacace R, et al.
Acta Neuropathol Commun
. 2015 Nov;
3:68.
PMID: 26555887
TAR DNA-binding protein 43 (TDP-43) inclusions are pathological hallmarks of patients with frontotemporal lobar degeneration (FTLD) and amyotrophic lateral sclerosis (ALS). Loss of TDP-43 in zebrafish engenders a severe muscle...
5.
Janssens J, Wils H, Kleinberger G, Joris G, Cuijt I, Ceuterick-de Groote C, et al.
Mol Neurobiol
. 2013 Mar;
48(1):22-35.
PMID: 23475610
Mutations in TAR DNA-binding protein 43 (TDP-43) are associated with familial forms of amyotrophic lateral sclerosis (ALS), while wild-type TDP-43 is a pathological hallmark of patients with sporadic ALS and...
6.
Wils H, Kleinberger G, Pereson S, Janssens J, Capell A, Van Dam D, et al.
J Pathol
. 2012 Jun;
228(1):67-76.
PMID: 22733568
Loss-of-function mutations in progranulin (GRN) are associated with frontotemporal lobar degeneration with intraneuronal ubiquitinated protein accumulations composed primarily of hyperphosphorylated TDP-43 (FTLD-TDP). The mechanism by which GRN deficiency causes TDP-43...
7.
Gijselinck I, Van Langenhove T, van der Zee J, Sleegers K, Philtjens S, Kleinberger G, et al.
Lancet Neurol
. 2011 Dec;
11(1):54-65.
PMID: 22154785
Background: Amyotrophic lateral sclerosis (ALS) and frontotemporal lobar degeneration (FTLD) are extremes of a clinically, pathologically, and genetically overlapping disease spectrum. A locus on chromosome 9p21 has been associated with...
8.
Gijselinck I, Engelborghs S, Maes G, Cuijt I, Peeters K, Mattheijssens M, et al.
Arch Neurol
. 2010 May;
67(5):606-16.
PMID: 20457961
Background: Frontotemporal lobar degeneration (FTLD) is a neurodegenerative brain disorder that can be accompanied by signs of amyotrophic lateral sclerosis (ALS). Objective: To identify a novel gene for FTLD-ALS. Design:...
9.
Wils H, Kleinberger G, Janssens J, Pereson S, Joris G, Cuijt I, et al.
Proc Natl Acad Sci U S A
. 2010 Feb;
107(8):3858-63.
PMID: 20133711
Neuronal cytoplasmic and intranuclear aggregates of RNA-binding protein TDP-43 are a hallmark feature of neurodegenerative diseases such as amyotrophic lateral sclerosis (ALS) and frontotemporal lobar degeneration (FTLD). ALS and FTLD...
10.
Pereson S, Wils H, Kleinberger G, McGowan E, Vandewoestyne M, Van Broeck B, et al.
J Pathol
. 2009 Jun;
219(2):173-81.
PMID: 19557827
Amyloid-beta (Abeta) plaques are pathological hallmarks of Alzheimer disease (AD). In addition, innate inflammatory responses, such as those mediated by microglia, are integral to the pathogenesis of AD. Interestingly, only...