Ibolya E Andras
Overview
Explore the profile of Ibolya E Andras including associated specialties, affiliations and a list of published articles.
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Articles
33
Citations
1074
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Recent Articles
1.
Andras I, Serrano N, Djuraskovic I, Fattakhov N, Sun E, Toborek M
Mol Neurobiol
. 2023 Jul;
60(11):6441-6465.
PMID: 37458985
Brain endothelial extracellular vesicles carrying amyloid beta (EV-Aβ) can be transferred to neural progenitor cells (NPCs) leading to NPC dysfunction. However, the events involved in this EV-mediated Aβ pathology are...
2.
Andras I, Serrano N, Djuraskovic I, Fattakhov N, Sun E, Toborek M
Res Sq
. 2023 Feb;
PMID: 36824983
Brain endothelial extracellular vesicles carrying amyloid beta (EV-Aβ) can be transferred to neural progenitor cells (NPCs) leading to NPC dysfunction. However, the events involved in this EV-mediated Aβ pathology are...
3.
Osborne O, Kowalczyk J, Louis K, Daftari M, Colbert B, Naranjo O, et al.
Extracell Vesicles Circ Nucl Acids
. 2023 Jan;
3(4):340-362.
PMID: 36649440
Aim: Elevated brain deposits of amyloid beta (Aβ) contribute to neuropathology and cognitive dysfunction in Alzheimer's disease (AD). However, the role of the blood-brain barrier (BBB) as an interface for...
4.
Cho H, Velichkovska M, Schurhoff N, Andras I, Toborek M
Neurobiol Dis
. 2021 May;
155:105388.
PMID: 33962010
Human immunodeficiency virus-1 (HIV-1) has been shown to cross the blood-brain barrier and cause HIV-associated neurocognitive disorders (HAND) through a process that may involve direct or indirect interactions with the...
5.
Andras I, Sewell B, Toborek M
Int J Mol Sci
. 2020 Apr;
21(8).
PMID: 32326569
Amyloid beta (Aβ) depositions are more abundant in HIV-infected brains. The blood-brain barrier, with its backbone created by endothelial cells, is assumed to be a core player in Aβ homeostasis...
6.
Andras I, Garcia-Contreras M, Yanick C, Perez P, Sewell B, Durand L, et al.
Mol Brain
. 2020 Feb;
13(1):21.
PMID: 32066471
Amyloid beta (Aβ) deposition was demonstrated to be elevated in the brains of HIV-infected patients and associated with neurocognitive decline; however, the mechanisms of these processes are poorly understood. The...
7.
Andras I, Leda A, Contreras M, Bertrand L, Park M, Skowronska M, et al.
Mol Cell Neurosci
. 2017 Jan;
79:12-22.
PMID: 28040512
HIV-infected brains are characterized by increased amyloid beta (Aβ) deposition. It is believed that the blood-brain barrier (BBB) is critical for Aβ homeostasis and contributes to Aβ accumulation in the...
8.
Andras I, Toborek M
Tissue Barriers
. 2016 May;
4(1):e1131804.
PMID: 27141419
Extracellular vesicles (ECV), like exosomes, gained recently a lot of attention as potentially playing a significant role in neurodegenerative diseases, particularly in Aβ pathology. While there are a lot of...
9.
Eum S, Jaraki D, Andras I, Toborek M
Toxicol Appl Pharmacol
. 2015 Jun;
287(3):258-66.
PMID: 26080028
Occludin is an essential integral transmembrane protein regulating tight junction (TJ) integrity in brain endothelial cells. Phosphorylation of occludin is associated with its localization to TJ sites and incorporation into...
10.
Transcriptional profile of HIV-induced nuclear translocation of amyloid β in brain endothelial cells
Andras I, Rampersaud E, Eum S, Toborek M
Arch Med Res
. 2014 Dec;
45(8):744-52.
PMID: 25446617
Background And Aims: Increased amyloid deposition in HIV-infected brains may contribute to the pathogenesis of neurocognitive dysfunction in infected patients. We have previously shown that exposure to HIV results in...