Helena C F Oliveira
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Explore the profile of Helena C F Oliveira including associated specialties, affiliations and a list of published articles.
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62
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866
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Recent Articles
1.
Castelli J, Raposo H, Navarro C, Lazaro C, Sartori M, Dalla Costa A, et al.
FASEB J
. 2025 Feb;
39(3):e70389.
PMID: 39924926
Susceptibility to obesity differs depending on the genetic background and housing temperatures. We have recently reported that CETP expressing female mice are leaner due to increased lipolysis, brown adipose tissue...
2.
Queiroz M, Lazaro C, Dos Santos L, Rentz T, Virgilio-da-Silva J, Moraes-Vieira P, et al.
Ecotoxicol Environ Saf
. 2024 Mar;
275:116254.
PMID: 38547729
Heavy metal exposure leads to multiple system dysfunctions. The mechanisms are likely multifactorial and involve inflammation and oxidative stress. The aim of this study was to evaluate markers and risk...
3.
Rentz T, Dorighello G, Dos Santos R, Barreto L, Freitas I, Lazaro C, et al.
Biomolecules
. 2023 Oct;
13(10).
PMID: 37892238
CETP activity reduces plasma HDL-cholesterol concentrations, a correlate of an increased risk of atherosclerotic events. However, our recent findings suggest that CETP expression in macrophages promotes an intracellular antioxidant state,...
4.
Lazaro C, Victorio J, Davel A, Oliveira H
Am J Physiol Heart Circ Physiol
. 2023 Aug;
325(3):H592-H600.
PMID: 37539470
Endothelial dysfunction is an early manifestation of atherosclerosis. The cholesteryl ester transfer protein (CETP) has been considered proatherogenic by reducing plasma HDL levels. However, CETP may exhibit cell- or tissue-specific...
5.
Dorighello G, Assis L, Rentz T, Morari J, Santana M, Passarelli M, et al.
Antioxidants (Basel)
. 2022 Sep;
11(9).
PMID: 36139808
Plasma cholesteryl ester transfer protein (CETP) activity diminishes HDL-cholesterol levels and thus may increase atherosclerosis risk. Experimental evidence suggests CETP may also exhibit anti-inflammatory properties, but local tissue-specific functions of...
6.
Navarro C, Francisco A, Figueira T, Ronchi J, Oliveira H, Vercesi A, et al.
Eur J Pharmacol
. 2022 Jan;
917:174750.
PMID: 35032488
The mechanisms by which a high-fat diet (HFD) promotes non-alcoholic fatty liver disease (NAFLD) appear to involve liver mitochondrial dysfunction and redox imbalance. The functional loss of the enzyme NAD(P)...
7.
Alberici L, Oliveira H
Antioxid Redox Signal
. 2021 Aug;
36(13-15):953-968.
PMID: 34409856
Altered plasma triglyceride metabolism and changes in dietary fatty acid types and levels are major contributors to the development of metabolic and cardiovascular diseases such as fatty liver disease, obesity,...
8.
Dorighello G, Rovani J, Paim B, Rentz T, Assis L, Vercesi A, et al.
J Atheroscler Thromb
. 2021 Jun;
29(6):825-838.
PMID: 34092712
Aim: Atherosclerosis is responsible for high morbidity and mortality rates around the world. Local arterial oxidative stress is involved in all phases of atherosclerosis development. Mitochondria is a relevant source...
9.
Wanschel A, Guizoni D, Lorza-Gil E, Salerno A, Paiva A, Dorighello G, et al.
Biomolecules
. 2021 Jan;
11(1).
PMID: 33430172
Endothelial dysfunction precedes atherosclerosis and is an independent predictor of cardiovascular events. Cholesterol levels and oxidative stress are key contributors to endothelial damage, whereas high levels of plasma high-density lipoproteins...
10.
Rentz T, Wanschel A, de Carvalho Moi L, Lorza-Gil E, de Souza J, Dos Santos R, et al.
Front Physiol
. 2020 Dec;
11:599379.
PMID: 33329050
An early event in atherogenesis is the recruitment and infiltration of circulating monocytes and macrophage activation in the subendothelial space. Atherosclerosis subsequently progresses as a unresolved inflammatory disease, particularly in...