Bruno A Paim
Overview
Explore the profile of Bruno A Paim including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
10
Citations
110
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Dorighello G, Rovani J, Paim B, Rentz T, Assis L, Vercesi A, et al.
J Atheroscler Thromb
. 2021 Jun;
29(6):825-838.
PMID: 34092712
Aim: Atherosclerosis is responsible for high morbidity and mortality rates around the world. Local arterial oxidative stress is involved in all phases of atherosclerosis development. Mitochondria is a relevant source...
2.
Dorighello G, Inada N, Paim B, Pardo-Andreu G, Vercesi A, Oliveira H
Cell Biol Int
. 2018 Feb;
42(6):747-753.
PMID: 29427465
Cardiovascular diseases are major causes of death worldwide. Beyond the classical cholesterol risk factor, other conditions such as oxidative stress are well documented to promote atherosclerosis. The Mangifera indica L....
3.
Dorighello G, Paim B, Catarina R Leite A, Vercesi A, Oliveira H
Exp Gerontol
. 2017 Feb;
109:47-50.
PMID: 28213051
Ageing and atherosclerosis are associated with oxidative stress. Mitochondrial redox function declines with ageing. Here we tested whether ageing LDL receptor knockout mice (LDLr) develop spontaneous atherosclerosis and whether mitochondrial...
4.
Dorighello G, Paim B, Kiihl S, Ferreira M, Catharino R, Vercesi A, et al.
Oxid Med Cell Longev
. 2015 Dec;
2016:7843685.
PMID: 26635912
Atherosclerosis has been associated with mitochondria dysfunction and damage. Our group demonstrated previously that hypercholesterolemic mice present increased mitochondrial reactive oxygen (mtROS) generation in several tissues and low NADPH/NADP+ ratio....
5.
Dorighello G, Rovani J, Luhman C, Paim B, Raposo H, Vercesi A, et al.
Br J Nutr
. 2013 Nov;
111(6):979-86.
PMID: 24176004
Different regimens of food restriction have been associated with protection against obesity, diabetes and CVD. In the present study, we hypothesised that food restriction would bring benefits to atherosclerosis- and...
6.
Alberici L, Paim B, Zecchin K, Mirandola S, Pestana C, Castilho R, et al.
Lipids Health Dis
. 2013 Jun;
12:87.
PMID: 23764148
Background: We have previously demonstrated that increased rates of superoxide generation by extra-mitochondrial enzymes induce the activation of the mitochondrial ATP-sensitive potassium channel (mitoK(ATP)) in the livers of hypertriglyceridemic (HTG)...
7.
Alberici L, Oliveira H, Paim B, Mantello C, Augusto A, Zecchin K, et al.
Free Radic Biol Med
. 2009 Aug;
47(10):1432-9.
PMID: 19703550
We have recently demonstrated that hypertriglyceridemic (HTG) mice present both elevated body metabolic rates and mild mitochondrial uncoupling in the liver owing to stimulated activity of the ATP-sensitive potassium channel...
8.
Pardo-Andreu G, Paim B, Castilho R, Velho J, Delgado R, Vercesi A, et al.
Pharmacol Res
. 2008 May;
57(5):332-8.
PMID: 18450471
Atherosclerosis is linked to a number of oxidative events ranging from low-density lipoprotein (LDL) oxidation to the increased production of intracellular reactive oxygen species (ROS). We have recently demonstrated that...
9.
Paim B, Velho J, Castilho R, Oliveira H, Vercesi A
Free Radic Biol Med
. 2007 Nov;
44(3):444-51.
PMID: 17991444
We have previously proposed that hypercholesterolemic LDL receptor knockout (k/o) mice mitochondria possess a lower antioxidant capacity due to a large consumption of reducing equivalents from NADPH to sustain high...
10.
Campos C, Paim B, Cosso R, Castilho R, Rottenberg H, Vercesi A
Cytometry A
. 2006 May;
69(6):515-23.
PMID: 16680678
Background: Cytochrome c release from mitochondria to cytosol is a hallmark of apoptosis and is used to characterize the mitochondria-dependent pathway of this type of cell death. Techniques currently used...