H Braley-Mullen
Overview
Explore the profile of H Braley-Mullen including associated specialties, affiliations and a list of published articles.
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Articles
82
Citations
528
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Recent Articles
1.
Fang Y, Sharp G, Yagita H, Braley-Mullen H
J Pathol
. 2008 Sep;
216(4):505-13.
PMID: 18810759
Granulomatous experimental autoimmune thyroiditis (G-EAT) is induced by mouse thyroglobulin (MTG)-sensitized splenocytes activated in vitro with MTG and IL-12. Thyroid lesions reach maximal severity 20 days after cell transfer, and...
2.
Fondal Jr W, Sampson C, Sharp G, Braley-Mullen H
J Interferon Cytokine Res
. 2001 Dec;
21(11):971-80.
PMID: 11747629
Mouse thyroglobulin (MuTg)-sensitized spleen cells activated in vitro with MuTg induce experimental autoimmune thyroiditis (EAT) in recipient mice with a thyroid infiltrate consisting primarily of lymphocytes. A more severe and...
3.
Braley-Mullen H, Chen K, Wei Y, Yu S
J Immunol
. 2001 Dec;
167(12):7111-8.
PMID: 11739533
Nearly 100% of NOD.H-2h4 mice develop spontaneous autoimmune thyroiditis (SAT) and produce anti-mouse thyroglobulin autoantibodies when they receive 0.05% NaI in their drinking water beginning at 8 wk of age....
4.
Wei Y, Chen K, Sharp G, Yagita H, Braley-Mullen H
J Immunol
. 2001 Nov;
167(11):6678-86.
PMID: 11714840
Granulomatous experimental autoimmune thyroiditis (G-EAT) is induced by mouse thyroglobulin-sensitized spleen cells activated in vitro with mouse thyroglobulin, anti-IL-2R, and IL-12. G-EAT lesions reach maximal severity 19-21 days after cell...
5.
Chen K, Wei Y, Sharp G, Braley-Mullen H
J Immunol
. 2001 Jul;
167(3):1720-7.
PMID: 11466396
Granulomatous experimental autoimmune thyroiditis (G-EAT) is induced by transfer of mouse thyroglobulin (MTg)-sensitized spleen cells activated in vitro with MTg and anti-IL-2R or MTg and IL-12. Previous work suggested that...
6.
Yu S, Medling B, Yagita H, Braley-Mullen H
J Autoimmun
. 2001 Feb;
16(1):37-46.
PMID: 11221995
Thyroid lesions develop in most NOD.H-2h4 mice 6 weeks after they are given 0.05% NaI in drinking water. B cells are required for spontaneous autoimmune thyroiditis (SAT) development, and anti-thyroglobulin...
7.
Chen K, Wei Y, Sharp G, Braley-Mullen H
J Leukoc Biol
. 2000 Dec;
68(6):828-35.
PMID: 11129650
This study was initiated to identify and characterize thyroid fibrosis in a murine model of granulomatous experimental autoimmune thyroiditis (G-EAT) and determine if TGF-beta1 might be involved in fibrosis. G-EAT...
8.
Braley-Mullen H, Sharp G
Int Rev Immunol
. 2000 Dec;
19(6):535-55.
PMID: 11129114
Experimental autoimmune thyroiditis (EAT) is a chronic inflammatory autoimmune disease that can be induced in genetically susceptible animals by immunization with mouse thyroglobulin (MTg) in an appropriate adjuvant or by...
9.
Braley-Mullen H, Yu S
J Immunol
. 2000 Dec;
165(12):7262-9.
PMID: 11120860
B cells are known to play an important role in the pathogenesis of several autoimmune diseases. NOD.H-2h4 mice develop spontaneous autoimmune thyroiditis (SAT) and anti-mouse thyroglobulin (MTg) autoantibodies, the levels...
10.
Tang H, Chen K, Wei Y, Sharp G, McKee L, Braley-Mullen H
Int Immunol
. 2000 Dec;
12(12):1629-39.
PMID: 11099302
Experimental autoimmune thyroiditis (EAT) with granulomatous histopathology (G-EAT) can be induced by cells from mouse thyroglobulin (MTg)-immunized donors activated in vitro with MTg and IL-12. G-EAT lesions reach maximum severity...