Gustavo A Rodriguez
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Explore the profile of Gustavo A Rodriguez including associated specialties, affiliations and a list of published articles.
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9
Citations
777
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Recent Articles
1.
Rodriguez G, Rothenberg E, Shetler C, Aoun A, Posani L, Vajram S, et al.
bioRxiv
. 2024 Dec;
PMID: 39651258
The progressive accumulation of amyloid beta (Aβ) pathology in the brain has been associated with aberrant neuronal network activity and poor cognitive performance in preclinical mouse models of Alzheimer's disease...
2.
Aoun A, Shetler O, Raghuraman R, Rodriguez G, Hussaini S
Front Cell Neurosci
. 2024 Feb;
17:1273283.
PMID: 38303974
Introduction: Spatial representations in the entorhinal cortex (EC) and hippocampus (HPC) are fundamental to cognitive functions like navigation and memory. These representations, embodied in spatial field maps, dynamically remap in...
3.
Aoun A, Shetler O, Raghuraman R, Rodriguez G, Hussaini S
bioRxiv
. 2023 Jul;
PMID: 37503011
Spatial representations in the entorhinal cortex (EC) and hippocampus (HPC) are fundamental to cognitive functions like navigation and memory. These representations, embodied in spatial field maps, dynamically remap in response...
4.
Rodriguez G, Barrett G, E Duff K, Hussaini S
PLoS Biol
. 2020 Aug;
18(8):e3000851.
PMID: 32822389
High levels of the amyloid-beta (Aβ) peptide have been shown to disrupt neuronal function and induce hyperexcitability, but it is unclear what effects Aβ-associated hyperexcitability may have on tauopathy pathogenesis...
5.
Fu H, Rodriguez G, Herman M, Emrani S, Nahmani E, Barrett G, et al.
Neuron
. 2017 Jan;
93(3):533-541.e5.
PMID: 28111080
The earliest stages of Alzheimer's disease (AD) are characterized by the formation of mature tangles in the entorhinal cortex and disorientation and confusion when navigating familiar places. The medial entorhinal...
6.
Wu J, Hussaini S, Bastille I, Rodriguez G, Mrejeru A, Rilett K, et al.
Nat Neurosci
. 2016 Jun;
19(8):1085-92.
PMID: 27322420
Tau protein can transfer between neurons transneuronally and trans-synaptically, which is thought to explain the progressive spread of tauopathy observed in the brain of patients with Alzheimer's disease. Here we...
7.
Rodriguez G, Tai L, LaDu M, Rebeck G
J Neuroinflammation
. 2014 Jun;
11:111.
PMID: 24948358
Background: Having the apolipoprotein E4 (APOE-ϵ4) allele is the strongest genetic risk factor for the development of Alzheimer's disease (AD). Accumulation of amyloid beta (Aβ) in the brain is influenced...
8.
Zhao W, Dumanis S, Tamboli I, Rodriguez G, LaDu M, Moussa C, et al.
Hum Mol Genet
. 2013 Oct;
23(5):1365-75.
PMID: 24154541
Intraneuronal accumulation of β-amyloid (Aβ)42 is one of the earliest pathological events in humans and in animal models of Alzheimer's disease (AD). Apolipoprotein E 4 (APOE4) is the major identified...
9.
Rodriguez G, Burns M, Weeber E, Rebeck G
Learn Mem
. 2013 Apr;
20(5):256-66.
PMID: 23592036
The apolipoprotein E4 (APOE-ε4) allele is the strongest genetic risk factor for developing late-onset Alzheimer's disease, and may predispose individuals to Alzheimer's-related cognitive decline by affecting normal brain function early...