Greet Vanderlinden
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Explore the profile of Greet Vanderlinden including associated specialties, affiliations and a list of published articles.
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11
Citations
36
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Recent Articles
1.
Vanderlinden G, Koole M, Michiels L, Lemmens R, Vandenbulcke M, Van Laere K
Alzheimers Dement
. 2024 Dec;
21(2):e14412.
PMID: 39732507
Introduction: The longitudinal progression of synaptic loss in Alzheimer's disease (AD) and how it is affected by tau pathology remains poorly understood. Methods: Thirty patients with amnestic mild cognitive impairment...
2.
Devulder A, Vanderlinden G, Cleeren E, Goovaerts V, Theys T, Van Laere K, et al.
BMC Neurol
. 2024 Dec;
24(1):478.
PMID: 39696024
Background: Neuronal hyperexcitability has been proposed to play a key role in Alzheimer's disease (AD). Understanding the relation between this enhanced excitability and AD pathology could provide a window for...
3.
Tang C, Vanderlinden G, Schroyen G, Deprez S, Van Laere K, Koole M
J Cereb Blood Flow Metab
. 2024 Oct;
45(3):568-577.
PMID: 39397394
A Support Vector Machine (SVM) based approach was developed to identify a pseudo-reference region for brain PET scans with the aim of reducing interscan and intersubject variability. By training a...
4.
Vande Casteele T, Laroy M, Van Cauwenberge M, Vanderlinden G, Vansteelandt K, Koole M, et al.
Am J Geriatr Psychiatry
. 2024 Aug;
33(1):47-62.
PMID: 39107144
Objective: To investigate whether tau accumulation is higher in late life depression (LLD) compared to non-depressed cognitively unimpaired (CU) older adults. To situate these findings in the neurodegeneration model of...
5.
Devulder A, Vanderlinden G, van Langenhoven L, Testelmans D, Van Den Bossche M, De Winter F, et al.
Brain
. 2024 Jul;
148(2):506-520.
PMID: 38990981
Both sleep alterations and epileptiform activity are associated with the accumulation of amyloid-β and tau pathology and are currently investigated for potential therapeutic interventions in Alzheimer's disease. However, a bidirectional...
6.
Wang J, Huang Q, He K, Li J, Guo T, Yang Y, et al.
Alzheimers Dement
. 2024 Apr;
20(6):3876-3888.
PMID: 38634334
Introduction: Metabotropic glutamate receptor 5 (mGluR5) is involved in regulating integrative brain function and synaptic transmission. Aberrant mGluR5 signaling and relevant synaptic failure play a key role in the pathophysiological...
7.
Vanderlinden G, Carron C, Vandenberghe R, Vandenbulcke M, Van Laere K
BMC Med Imaging
. 2024 Feb;
24(1):41.
PMID: 38347458
Background: F-FDG brain PET is clinically used for differential diagnosis in cognitive dysfunction of unclear etiology and for exclusion of a neurodegenerative cause in patients with cognitive impairment in late-life...
8.
Vanderlinden G, Michiels L, Koole M, Lemmens R, Liessens D, Van Walleghem J, et al.
J Neurotrauma
. 2023 Dec;
41(3-4):420-429.
PMID: 38038357
Epidemiological studies have identified prior traumatic brain injury (TBI) as a risk factor for developing Alzheimer's disease (AD). Neurofibrillary tangles (NFTs) are common to AD and chronic traumatic encephalopathy following...
9.
Vanderlinden G, Mertens N, Michiels L, Lemmens R, Koole M, Vandenbulcke M, et al.
Eur J Nucl Med Mol Imaging
. 2022 Jul;
49(13):4580-4588.
PMID: 35852556
Purpose: Neurofibrillary tangles (NFTs) in Alzheimer's disease can be accurately quantified in vivo using [F]MK-6240 PET. Short-term [F]MK-6240 test-retest (TRT) is about 6%, but also long-term stability of cerebral uptake...
10.
Vanderlinden G, Ceccarini J, Vande Casteele T, Michiels L, Lemmens R, Triau E, et al.
Mol Psychiatry
. 2022 Jul;
27(10):4244-4251.
PMID: 35794185
Next to amyloid and tau, synaptic loss is a key pathological hallmark in Alzheimer's disease, closely related to cognitive dysfunction and neurodegeneration. Tau is thought to cause synaptic loss, but...