Glenna J Peterson
Overview
Explore the profile of Glenna J Peterson including associated specialties, affiliations and a list of published articles.
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Articles
17
Citations
114
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Recent Articles
1.
Dill-McFarland K, Simmons J, Peterson G, Nguyen F, Campo M, Benchek P, et al.
mSystems
. 2024 Aug;
9(9):e0062824.
PMID: 39162406
(Mtb) exposure leads to a range of outcomes including clearance, latent TB infection (LTBI), and pulmonary tuberculosis (TB). Some heavily exposed individuals resist tuberculin skin test (TST) and interferon-gamma (IFNγ)...
2.
Campo M, Dill-McFarland K, Peterson G, Benson B, Skerrett S, Hawn T
J Immunol
. 2024 Jun;
213(2):161-169.
PMID: 38836816
Alveolar macrophages (AMs) and recruited monocyte-derived macrophages (MDMs) mediate early lung immune responses to Mycobacterium tuberculosis. Differences in the response of these distinct cell types are poorly understood and may...
3.
Hong H, Dill-McFarland K, Simmons J, Peterson G, Benchek P, Mayanja-Kizza H, et al.
Front Immunol
. 2024 Mar;
15:1359178.
PMID: 38515745
Introduction: The heterogeneity of outcomes after (Mtb) exposure is a conundrum associated with millennia of host-pathogen co-evolution. We hypothesized that human myeloid cells contain genetically encoded, Mtb-specific responses that regulate...
4.
Dill-McFarland K, Simmons J, Peterson G, Nguyen F, Campo M, Benchek P, et al.
bioRxiv
. 2024 Mar;
PMID: 38464296
(Mtb) exposure leads to a range of outcomes including clearance, latent TB infection (LTBI), and pulmonary tuberculosis (TB). Some heavily exposed individuals resist tuberculin skin test (TST) and interferon gamma...
5.
Hong H, Dill-McFarland K, Benson B, Simmons J, Peterson G, Benchek P, et al.
AIDS
. 2023 Sep;
37(15):2287-2296.
PMID: 37696247
Objective: To determine whether Mycobacterium tuberculosis (Mtb)-induced monocyte transcriptional responses differ in people with HIV (PWH) who do (RSTR) or do not (LTBI) resist tuberculin skin test/interferon-γ (IFN-γ) release assay...
6.
Hong H, Dill-McFarland K, Simmons J, Peterson G, Benchek P, Mayanja-Kizza H, et al.
medRxiv
. 2023 Sep;
PMID: 37693490
The heterogeneity of outcomes after (Mtb) exposure is a conundrum associated with millennia of host-pathogen co-evolution. We hypothesized that human myeloid cells contain genetically encoded, Mtb-specific responses that regulate critical...
7.
Simmons J, Segnitz R, Dill-McFarland K, Stein C, Peterson G, Mayanja-Kizza H, et al.
PLoS One
. 2023 Apr;
18(4):e0284498.
PMID: 37058459
Background: A mechanistic understanding of uncommon immune outcomes such as resistance to infection has led to the development of novel therapies. Using gene level analytic methods, we previously found distinct...
8.
Arlehamn C, Benson B, Kuan R, Dill-McFarland K, Peterson G, Paul S, et al.
Front Immunol
. 2022 Oct;
13:1016038.
PMID: 36263044
Immunological mechanisms of susceptibility to nontuberculous mycobacterial (NTM) disease are poorly understood. To understand NTM pathogenesis, we evaluated innate and antigen-specific adaptive immune responses to complex (MAC) in asymptomatic individuals...
9.
Simmons J, Dill-McFarland K, Stein C, Van P, Chihota V, Ntshiqa T, et al.
mSphere
. 2022 Jun;
7(3):e0015922.
PMID: 35695527
Heavy exposure to Mycobacterium tuberculosis, the etiologic agent of tuberculosis (TB) and among the top infectious killers worldwide, results in infection that is cleared, contained, or progresses to disease. Some...
10.
Simmons J, Van P, Stein C, Chihota V, Ntshiqa T, Maenetje P, et al.
J Clin Invest
. 2021 Jun;
131(14).
PMID: 34111032
After extensive exposure to Mycobacterium tuberculosis (Mtb), most individuals acquire latent Mtb infection (LTBI) defined by a positive tuberculin skin test (TST) or interferon-γ release assay (IGRA). To identify mechanisms...