Georges Levesque
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Explore the profile of Georges Levesque including associated specialties, affiliations and a list of published articles.
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Citations
253
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Recent Articles
1.
Huang F, Ben Aissa M, Levesque G, Carreau M
BMC Res Notes
. 2018 Sep;
11(1):662.
PMID: 30213274
Objective: The Uncoordinated 5A (UNC5A) protein is part of a family of receptors that play roles in axonal pathfinding and cell migration. We previously showed that the Fanconi anemia C...
2.
Tremblay C, Huang F, Levesque G, Carreau M
BMC Res Notes
. 2018 Feb;
11(1):138.
PMID: 29463306
Objective: The Hairy Enhancer of Split 1 (HES1) is a transcriptional repressor that regulates cellular proliferation and differentiation during development. We previously found an interaction between HES1 and Fanconi anemia...
3.
Huang F, Ben Aissa M, Magron A, C Huard C, Godin C, Levesque G, et al.
PLoS One
. 2014 Mar;
9(3):e92811.
PMID: 24676280
The Fanconi anemia group C protein (FANCC) is one of the several proteins that comprise the Fanconi anemia (FA) network involved in genomic surveillance. FANCC is mainly cytoplasmic and has...
4.
C Huard C, Tremblay C, Magron A, Levesque G, Carreau M
Proc Natl Acad Sci U S A
. 2014 Jan;
111(6):2152-7.
PMID: 24469828
Fanconi anemia (FA) is an inherited bone marrow failure syndrome associated with a progressive decline in hematopoietic stem cells, developmental defects, and predisposition to cancer. These various phenotypic features imply...
5.
C Huard C, Tremblay C, Helsper K, Delisle M, Schindler D, Levesque G, et al.
Blood
. 2013 Jan;
121(10):1729-39.
PMID: 23303816
Fanconi anemia (FA) is a genetic disorder characterized by congenital abnormalities, bone marrow failure, and increased susceptibility to cancer. Of the fifteen FA proteins, Fanconi anemia group C (FANCC) is...
6.
Jun G, Moncaster J, Koutras C, Seshadri S, Buros J, McKee A, et al.
PLoS One
. 2012 Sep;
7(9):e43728.
PMID: 22984439
Multiple lines of evidence suggest that specific subtypes of age-related cataract (ARC) and Alzheimer disease (AD) are related etiologically. To identify shared genetic factors for ARC and AD, we estimated...
7.
Bretteville A, Marcouiller F, Julien C, Khoury N, Petry F, Poitras I, et al.
Sci Rep
. 2012 Jul;
2:480.
PMID: 22761989
Tau hyperphosphorylation is one hallmark of Alzheimer's disease (AD) pathology. Pharmaceutical companies have thus developed kinase inhibitors aiming to reduce tau hyperphosphorylation. One obstacle in screening for tau kinase inhibitors...
8.
Ben Aissa M, April M, Bergeron L, Perreault J, Levesque G
Int J Alzheimers Dis
. 2012 Apr;
2012:947147.
PMID: 22482079
Alzheimer's disease (AD) etiological studies suggest that an elevation in amyloid-β peptides (Aβ) level contributes to aggregations of the peptide and subsequent development of the disease. The major constituent of...
9.
Koutras C, Levesque G
PLoS One
. 2011 Oct;
6(10):e25379.
PMID: 22022388
Neural plakophilin-related armadillo protein (NPRAP or δ-catenin) is a neuronal-specific protein that is best known for its interaction with presenilin 1 (PS1). Interestingly, the hemizygous loss of NPRAP is associated...
10.
Koutras C, Lessard C, Levesque G
J Alzheimers Dis
. 2011 Aug;
27(2):307-16.
PMID: 21811021
Presenilin-1 (PS1) is a broadly expressed transmembrane protein that is often mutated in familial Alzheimer's disease (AD). In addition to its role in amyloid production, PS1 interacts with several protein...