G Szabadkai
Overview
Explore the profile of G Szabadkai including associated specialties, affiliations and a list of published articles.
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Articles
28
Citations
1698
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Recent Articles
1.
Llorente-Folch I, Rueda C, Pardo B, Szabadkai G, Duchen M, Satrustegui J
J Physiol
. 2015 Mar;
593(16):3447-62.
PMID: 25809592
Calcium signalling is fundamental to the function of the nervous system, in association with changes in ionic gradients across the membrane. Although restoring ionic gradients is energetically costly, a rise...
2.
Galluzzi L, Bravo-San Pedro J, Vitale I, Aaronson S, Abrams J, Adam D, et al.
Cell Death Differ
. 2014 Sep;
22(1):58-73.
PMID: 25236395
Cells exposed to extreme physicochemical or mechanical stimuli die in an uncontrollable manner, as a result of their immediate structural breakdown. Such an unavoidable variant of cellular demise is generally...
3.
Galluzzi L, Vitale I, Michels J, Brenner C, Szabadkai G, Harel-Bellan A, et al.
Cell Death Dis
. 2014 May;
5:e1257.
PMID: 24874729
The platinum derivative cis-diamminedichloroplatinum(II), best known as cisplatin, is currently employed for the clinical management of patients affected by testicular, ovarian, head and neck, colorectal, bladder and lung cancers. For...
4.
Dongworth R, Mukherjee U, Hall A, Astin R, Ong S, Yao Z, et al.
Cell Death Dis
. 2014 Mar;
5:e1082.
PMID: 24577080
Novel therapeutic targets are required to protect the heart against cell death from acute ischemia-reperfusion injury (IRI). Mutations in the DJ-1 (PARK7) gene in dopaminergic neurons induce mitochondrial dysfunction and...
5.
Tufo G, Jones A, Wang Z, Hamelin J, Tajeddine N, Esposti D, et al.
Cell Death Differ
. 2014 Jan;
21(5):685-95.
PMID: 24464223
Intrinsic and acquired chemoresistance are frequent causes of cancer eradication failure. Thus, long-term cis-diaminedichloroplatine(II) (CDDP) or cisplatin treatment is known to promote tumor cell resistance to apoptosis induction via multiple...
6.
Yao Z, Jones A, Fassone E, Sweeney M, Lebiedzinska M, Suski J, et al.
Oncogene
. 2012 Jul;
32(20):2592-600.
PMID: 22777349
Primary mitochondrial dysfunction commonly leads to failure in cellular adaptation to stress. Paradoxically, however, nonsynonymous mutations of mitochondrial DNA (mtDNA) are frequently found in cancer cells and may have a...
7.
Traba J, Del Arco A, Duchen M, Szabadkai G, Satrustegui J
Cell Death Differ
. 2011 Oct;
19(4):650-60.
PMID: 22015608
Ca(2+)-mediated mitochondrial permeability transition (mPT) is the final common pathway of stress-induced cell death in many major pathologies, but its regulation in intact cells is poorly understood. Here we report...
8.
Skipworth J, Szabadkai G, Olde Damink S, Leung P, Humphries S, Montgomery H
Aliment Pharmacol Ther
. 2011 Aug;
34(8):840-52.
PMID: 21851372
Background: In addition to the circulating (endocrine) renin-angiotensin system (RAS), local renin-angiotensin systems are now known to exist in diverse cells and tissues. Amongst these, pancreatic renin-angiotensin systems have recently...
9.
Vicencio J, Estrada M, Galvis D, Bravo R, Contreras A, Rotter D, et al.
Mini Rev Med Chem
. 2011 Mar;
11(5):390-8.
PMID: 21443511
Increasing evidence suggests that nongenomic effects of testosterone and anabolic androgenic steroids (AAS) operate concertedly with genomic effects. Classically, these responses have been viewed as separate and independent processes, primarily...
10.
Vicencio J, Ortiz C, Criollo A, Jones A, Kepp O, Galluzzi L, et al.
Cell Death Differ
. 2009 Mar;
16(7):1006-17.
PMID: 19325567
The inositol 1,4,5-trisphosphate receptor (IP(3)R) is a major regulator of apoptotic signaling. Through interactions with members of the Bcl-2 family of proteins, it drives calcium (Ca(2+)) transients from the endoplasmic...