Franziska V Kraus
Overview
Explore the profile of Franziska V Kraus including associated specialties, affiliations and a list of published articles.
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5
Citations
65
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Recent Articles
1.
Chakraborty S, Gupta R, Kubatzky K, Kar S, Kraus F, Souto-Carneiro M, et al.
Clin Immunol
. 2023 Oct;
257:109814.
PMID: 37879380
In Rheumatoid Arthritis (RA), regulatory T cells (Tregs) have been found to be enriched in the synovial fluid. Despite their accumulation, they are unable to suppress synovial inflammation. Recently, we...
2.
Seebach E, Kraus F, Elschner T, Kubatzky K
Inflamm Res
. 2023 Jun;
72(7):1465-1484.
PMID: 37329360
Introduction: The pathophysiology of chronic implant-related bone infections is characterized by an increase in osteoclast numbers and enhanced bone resorption. Biofilms are a major reason for chronicity of such infections...
3.
Seebach E, Elschner T, Kraus F, Souto-Carneiro M, Kubatzky K
Inflammation
. 2023 May;
46(4):1512-1530.
PMID: 37212952
Biofilm formation is a leading cause for chronic implant-related bone infections as biofilms shield bacteria against the immune system and antibiotics. Additionally, biofilms generate a metabolic microenvironment that shifts the...
4.
Kraus F, Keck S, Klika K, Graf J, Carvalho R, Lorenz H, et al.
Arthritis Rheumatol
. 2023 Jan;
75(7):1098-1109.
PMID: 36704915
Objective: Rheumatoid arthritis (RA) CD8+ T cells maintain their effector proinflammatory phenotype by changing their metabolism toward aerobic glycolysis. However, their massive energy and biosynthesis needs may require additional substrates...
5.
Souto-Carneiro M, Klika K, Abreu M, Meyer A, Saffrich R, Sandhoff R, et al.
Arthritis Rheumatol
. 2020 Jul;
72(12):2050-2064.
PMID: 32602217
Objective: CD8+ T cells contribute to rheumatoid arthritis (RA) by releasing proinflammatory and cytolytic mediators, even in a challenging hypoxic and nutrient-poor microenvironment such as the synovial membrane. This study...